Anatomy & Physiology
3rd Edition
ISBN: 9781259398629
Author: McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa Stouter
Publisher: Mcgraw Hill Education,
expand_more
expand_more
format_list_bulleted
Concept explainers
Question
Chapter 15, Problem 1CSL
Summary Introduction
To determine:
The necessity of having an expensive mechanism of mass activation response of the sympathetic nervous system at disposal.
Concept introduction:
The sympathetic nervous system is most active when something exciting is going to happen. Mass activation of the sympathetic nervous system refers to the stimulation of various body structures through sympathetic neurons. Mass activation is often a response to a crisis.
Expert Solution & Answer
Want to see the full answer?
Check out a sample textbook solutionStudents have asked these similar questions
Which of the following statements is true?
Chemical messengers within cells are always the main determinant of the effector response.
Dual innervation is always present in all organs and glands; one branch enhances the function or secretion, while the other branch inhibits it.
The beating of the heart is regulated only by the sympathetic division.
Some blood vessels contain alpha adrenergic receptors that cause vasoconstriction in the presence of epinephrine, whereas others have beta adrenergic receptors that cause vasodilation in the presence of epinephrine.
The drug Celebrex selectively inhibits PTGS2 while aspirin and other NSAID’s inhibit both PTGS1 and PTGS2 in the same way. Why do you think researchers wanted to discover a selective inhibitor to PTGS2?
Halle Zingiber has been granted refugee status in this country. He has had to leave his wife and two children in Ethiopia. He is severely depressed and is receiving treatment with the tricyclic depressant, nortriptyline. No effects were reported when standard doses of the drug (25 mg tds) were used. His doctor increased the dose many-fold to 200 mg/day a dose, which is inducing effects. Can you explain Halle’s unusual response to therapy?
Chapter 15 Solutions
Anatomy & Physiology
Ch. 15.1 - LEARNING OBJECTIVE
1. List the similarities and...Ch. 15.1 - What criterion is used to organize the nervous...Ch. 15.1 - Prob. 2LOCh. 15.1 - Prob. 3LOCh. 15.1 - Prob. 2WDLCh. 15.1 - Prob. 4LOCh. 15.1 - What CNS structure is the integration and command...Ch. 15.2 - Prob. 5LOCh. 15.2 - Prob. 4WDLCh. 15.2 - Prob. 6LO
Ch. 15.2 - Describe the general anatomic differences in the...Ch. 15.2 - Prob. 7LOCh. 15.2 - Prob. 6WDLCh. 15.3 - Prob. 8LOCh. 15.3 - Prob. 1WDTCh. 15.3 - Which four cranial nerves have a parasympathetic...Ch. 15.3 - Prob. 9LOCh. 15.3 - What organs are innervated by the pelvic...Ch. 15.4 - Prob. 10LOCh. 15.4 - Prob. 11LOCh. 15.4 - Prob. 12LOCh. 15.4 - Prob. 13LOCh. 15.4 - Prob. 9WDLCh. 15.4 - Prob. 10WDLCh. 15.4 - Prob. 14LOCh. 15.4 - Prob. 15LOCh. 15.4 - Prob. 2WDTCh. 15.4 - Prob. 11WDLCh. 15.4 - In what ways does the adrenal medulla pathway help...Ch. 15.5 - Prob. 16LOCh. 15.5 - Prob. 17LOCh. 15.5 - Prob. 13WDLCh. 15.5 - Prob. 14WDLCh. 15.6 - Prob. 18LOCh. 15.6 - Prob. 15WDLCh. 15.6 - Prob. 19LOCh. 15.6 - Prob. 3WDTCh. 15.6 - Prob. 16WDLCh. 15.6 - Prob. 17WDLCh. 15.6 - Prob. 20LOCh. 15.6 - Prob. 21LOCh. 15.6 - Prob. 18WDLCh. 15.6 - Prob. 19WDLCh. 15.7 - LEARNING OBJECTIVE
22. Discuss the nature of...Ch. 15.7 - Prob. 20WDLCh. 15.7 - Prob. 23LOCh. 15.7 - LEARNING OBJECTIVE
24. Describe the antagonistic...Ch. 15.7 - Prob. 21WDLCh. 15.7 - Prob. 22WDLCh. 15.7 - Prob. 25LOCh. 15.7 - What are the body structures innervated by the...Ch. 15.8 - Prob. 26LOCh. 15.8 - 27. Describe some major examples of autonomic...Ch. 15.8 - Prob. 24WDLCh. 15 - A splanchnic nerve in the sympathetic division of...Ch. 15 - Some parasympathetic preganglionic neuron cell...Ch. 15 - Prob. 3DYBCh. 15 - Prob. 4DYBCh. 15 - Sympathetic division preganglionic axons travel to...Ch. 15 - Prob. 6DYBCh. 15 - Prob. 7DYBCh. 15 - A sympathetic postganglionic axon is a. long and...Ch. 15 - Prob. 9DYBCh. 15 - Prob. 10DYBCh. 15 - Prob. 11DYBCh. 15 - For the following ganglia, identify the location...Ch. 15 - Prob. 13DYBCh. 15 - Prob. 14DYBCh. 15 - Prob. 15DYBCh. 15 - Prob. 16DYBCh. 15 - Prob. 17DYBCh. 15 - What may occur with the mass activation of the...Ch. 15 - Prob. 19DYBCh. 15 - Prob. 20DYBCh. 15 - Prob. 1CALCh. 15 - Arlene was in a heightened state of alertness well...Ch. 15 - George has hypertension (high blood pressure). His...Ch. 15 - Prob. 4CALCh. 15 - Prob. 5CALCh. 15 - Prob. 1CSLCh. 15 - When you were younger, your parents may have told...Ch. 15 - Prob. 3CSL
Knowledge Booster
Learn more about
Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- Catecholamines are neurotransmitters that are produced in the adrenal glands and are released during a stress response. Some typical effects are increased heart rate, increased blood glucose levels and elevated blood pressure. Examples include dopamine, norepinephrine, and epinephrine. Reserpine is a drug that can control high blood pressure by reducing the number of catecholamine neurotransmitters present in the synapse. Which of the following rows correctly identifies the effect of reserpine in the synapse and the division of the nervous system it affects? Select one: а. Effect in the Synapse Division of the Nervous System Keeps catecholamines longer in the synapse Sympathetic nervous system b. Effect in the Synapse Division of the Nervous System Inhibits release of catecholamines from the presynaptic neuron Sympathetic nervous system С. Effect in the Synapse Division of the Nervous System Inhibits release of catecholamines from presynaptic neuron Parasympathetic nervous system d.…arrow_forwardCatecholamines are neurotransmitters that are produced in the adrenal glands and are released during a stress response. Some typical effects are increased heart rate, increased blood glucose levels and elevated blood pressure. Examples include dopamine, norepinephrine, and epinephrine. Reserpine is a drug that can control high blood pressure by reducing the number of catecholamine neurotransmitters present in the synapse. Which f the following rows correctly identifies the effect of reserpine in the synapse and the division of the nervous system it affects? Select one: Effect in the Synapse Division of the Nervous System Keeps catecholamines longer Sympathetic nervous system in the synapse b. Effect in the Synapse Division of the Nervous System Inhibits release of catecholamines from the presynaptic neuron Sympathetic nervous system Division of the Nervous System Effect in the Synapse Inhibits release of catecholamines from presynaptic neuron Parasympathetic nervous system d. Effect in…arrow_forwardThe sympathetic nervous system has a key function in the stress response. What is that?arrow_forward
- We talked about drug effects on neurons in sequence. The effect of alcohol is multi-faceted and the following question asks you to apply your knowledge. Imagine two neurons in sequence. The presynaptic neuron is GABAnergic and the postsynaptic neuron is dopaminergic. The effects of alcohol are not fully understood but it does seem to inhibit GABAnergic neurons. How would the release of dopamine from the postsynaptic neuron change in this case? Explain your answer, being sure to make each connection between concepts clear. If alcohol instead inhibited dopaminernergic neurons, in what way might the ion flow change in the dendrites of the postsynaptic neuron of this example?arrow_forwardBotulinum toxin (Botox) works by blocking the exocytosis of acetylcholine. A number of different medical and cosmetic procedures use this chemical. Injection of small quantities of botulinum toxin (Botox) into specific overactive muscles causes muscle relaxation that smooths the skin and reduces wrinkles. Injection into certain spinal ganglia helps reduce a certain form of chronic pain, and injections into the salivary glands causes the reduction of overactive saliva production. What type of toxin is Botox? a. Agonist b. Antagonistarrow_forwardBotulinum toxin (Botox) works by blocking the exocytosis of acetylcholine. A number of different medical and cosmetic procedures use this chemical. Injection of small quantities of botulinum toxin (Botox) into specific overactive muscles causes muscle relaxation that smooths the skin and reduces wrinkles. Injection into certain spinal ganglia helps reduce a certain form of chronic pain, and injections into the salivary glands causes the reduction of overactive saliva production. What type of toxin is Botox? Agonist or Antagonistarrow_forward
- When the neurotransmitter acetylcholine (ACh) binds the acetylcholine receptor (a GPCR) on muscle cells, it causes them to contract. ZIGGY, a chemical analog of ACh, also binds to the same acetylcholine receptor on muscle cells, but instead causes the muscle cells to relax. For this reason, it is sometimes prescribed as a muscle relaxer. Explain in 3-4 sentences how ZIGGY could cause muscle relaxation. How can both ZIGGY and ACh bind the same GPCR? And then how can they have different effects on the cells, despite binding to the same receptor on the same cells?arrow_forwardAcetylcholine is a neurotransmitter that, when bound to its receptor, causes the receptor to open a channel that allows the flow of Na+ ions into neurons. When the Na+ ions begin to flow into the neuron, this change in the net charge across the neuronal cell membrane often triggers Ca2+ ion channels to open. In this scenario, the acetylcholine receptor would be acting as a channel, and the Ca2+ channels would be classified as voltage-gated; leaky ligand gated; voltage-gated symporter; antiporter None of the above. ionarrow_forwardWhich statement is true regarding the nervous system? a) Damaged myelin on an axon has no effect with its ability to transmit an action potential b) Depolarization is the reduction of the membrane potential; going from negative to a more positive charge c) Neuroglia cells are only responsible for providing immunity to the nervous cells d) Neurons can communicate with other cells without the use of neurotransmittersarrow_forward
- A patient has been exposed to the organophosphate pesticide malathion,which inactivates acetylcholinesterase. Which of the following symptoms would you predict: blurring of vision, excess tear formation, frequent or involuntary urination, pallor (pale skin), muscle twitching, orcramps? Would atropine be an effective drug to treat the symptoms?(See Clinical Impact 16.2 for the action of atropine.) Explain.arrow_forwardThe fight-or-flight reaction prepares an animal to Pesponu Lu a threatening situation, by either fighting or running away. One aspect of this response is changes in blood flow such that the skeletal muscles and heart receive higher blood flow (vessels dilate) while digestive organs receive less (vessels constrict). A) Which signal molecule(s) is/are involved in this response, and how do they produce these changes in blood vessels? Briefly explain how this example illustrates how different cells are able to use the same ligands in different ways.arrow_forwardI am confused about the process of rod cells in the dark. In the dark, rod cells are unstimulated and therefore the sodium is able to enter and depolarize the cell, stimulating the release of glutamate to the bipolar cell. However, this increase in glutamate in the bipolar cell inhibits the release of action potential from the bipolar cell to the ganglion cell. This doesn't make sense to me because shouldn't there be an increase in action potential since the rod cells are supposed to function in the dark?arrow_forward
arrow_back_ios
SEE MORE QUESTIONS
arrow_forward_ios
Recommended textbooks for you
- Human Anatomy & Physiology (11th Edition)BiologyISBN:9780134580999Author:Elaine N. Marieb, Katja N. HoehnPublisher:PEARSONBiology 2eBiologyISBN:9781947172517Author:Matthew Douglas, Jung Choi, Mary Ann ClarkPublisher:OpenStaxAnatomy & PhysiologyBiologyISBN:9781259398629Author:McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa StouterPublisher:Mcgraw Hill Education,
- Molecular Biology of the Cell (Sixth Edition)BiologyISBN:9780815344322Author:Bruce Alberts, Alexander D. Johnson, Julian Lewis, David Morgan, Martin Raff, Keith Roberts, Peter WalterPublisher:W. W. Norton & CompanyLaboratory Manual For Human Anatomy & PhysiologyBiologyISBN:9781260159363Author:Martin, Terry R., Prentice-craver, CynthiaPublisher:McGraw-Hill Publishing Co.Inquiry Into Life (16th Edition)BiologyISBN:9781260231700Author:Sylvia S. Mader, Michael WindelspechtPublisher:McGraw Hill Education
Human Anatomy & Physiology (11th Edition)
Biology
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:PEARSON
Biology 2e
Biology
ISBN:9781947172517
Author:Matthew Douglas, Jung Choi, Mary Ann Clark
Publisher:OpenStax
Anatomy & Physiology
Biology
ISBN:9781259398629
Author:McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa Stouter
Publisher:Mcgraw Hill Education,
Molecular Biology of the Cell (Sixth Edition)
Biology
ISBN:9780815344322
Author:Bruce Alberts, Alexander D. Johnson, Julian Lewis, David Morgan, Martin Raff, Keith Roberts, Peter Walter
Publisher:W. W. Norton & Company
Laboratory Manual For Human Anatomy & Physiology
Biology
ISBN:9781260159363
Author:Martin, Terry R., Prentice-craver, Cynthia
Publisher:McGraw-Hill Publishing Co.
Inquiry Into Life (16th Edition)
Biology
ISBN:9781260231700
Author:Sylvia S. Mader, Michael Windelspecht
Publisher:McGraw Hill Education
Immune System and Immune Response Animation; Author: Medical Sciences Animations;https://www.youtube.com/watch?v=JDdbUBXPKc4;License: Standard YouTube License, CC-BY
Immune response: summary; Author: Dr Bhavsar Biology;https://www.youtube.com/watch?v=ADANgHkX4OY;License: Standard Youtube License