You are developing a anti-viral treatment for SARS-CoV-2. You decide to have your product target the replication cycle. What step would you target? Why would you target this step? HOW wold you target this step?

Why is HIV protease a good target for anti-HIV therapy and drug discovery?

Which step(s) in the replication cycle of HIV can still occur in the presence of a reverse transcriptase inhibitor? Select all that apply. transcription of viral genes initial viral attachment viral assembly integration of viral DNA into host genome initial viral entry

Tat and Rev proteins are required for HIV replication. What are their roles and why are they essential to the virus?

Can someone help me with my virology homework? Can you explain how to interpret these three figures?

The omicron variant of SARS-CoV-2 has the amino acid substitution G339D in the spike protein on the virus surface. In cellular fluids at -pH 7 the wild type spike protein has a net negative charge. This substitution will make the spike protein: less negative absorb UV light more strongly at 280 nm more negative more hydrophobic

Reverse transcriptase is an error prone DNA polymerase.   Describe the role of reverse transcriptase in viral propagation and how having an error-prone enzyme might make it challenging to have a vaccine

Please help explain this LAPTM5 restricts HIV-1 and Vpr counteracts the restriction

answer doesn't have to be long i just need an explanation, thank you!   The whole world right now is concerned (or they should be) about the appearance of new variants of SARS-CoV-2. Among other things, these variant viruses have mutations that change the amino acid sequence of the spike protein on the virus surface. While this is concerning, I would like to propose that there is a limit to how far these mutations can change the spike protein. Why can't there be an endless number of new mutations of this viral protein? How would these amino acid changes affect how the virus works?