Wich of the following can cause edema? A_ decrease in plasma proteins. B_ damage to capillart walls. 3 _ increased capillary hydrostaic pressure. F_ cirrhosis, heart failure,kidney desease. G. All the bove
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Wich of the following can cause edema?
A_ decrease in plasma proteins.
B_ damage to capillart walls.
3 _ increased capillary hydrostaic pressure.
F_ cirrhosis, heart failure,kidney desease.
G. All the bove
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- 1. angiotesnin II causes sympathetic blood pressure to increase decrese stay the same 2. in response to a decrease of blood pressure, the diameter of afferent arteriole to will increase decrese stay the same 3. as filtrate travels from the capsular space to the loop of henle, its glucose content will? increase decrese stay the sameA patient (76 years old female) with compromised cardiac function was hospitalized due to significant volume overload and generalized edema. Chest X-ray confirmed pulmonary edema, ejection fraction was estimated - 20%. Patients’ heart rate and blood pressure have been monitored. Despite intravenous use of diuretics she still feels significant shortness of breath, her blood pressure is now 90/60 mm Hg. Blood tests confirmed that renal function is worsening. Blood creatinine – 2.0 mg/L, Na – 132 mEq/L; K – 3.9 mEq/L. Intravenous therapy with Dobutamine was initiated. After intensive treatment at the hospital for more than a week, this patient was released home and will be followed by primary care physician. Along other medication lisinopril was prescribed. Questions: What group of cardiovascular drugs does lisinopril belong to? What is mechanism of action? What effect this drug has on patient’s neurohumoral responses? What potential side effects can be related to long term lisinopril…A patient (76 years old female) with compromised cardiac function was hospitalized due to significant volume overload and generalized edema. Chest X-ray confirmed pulmonary edema, ejection fraction was estimated - 20%. Patients’ heart rate and blood pressure have been monitored. Despite intravenous use of diuretics she still feels significant shortness of breath, her blood pressure is now 90/60 mm Hg. Blood tests confirmed that renal function is worsening. Blood creatinine – 2.0 mg/L, Na – 132 mEq/L; K – 3.9 mEq/L. Intravenous therapy with Dobutamine was initiated. Questions: What group of cardiovascular drugs does dobutamine belong to? What is mechanism of action? What effect this drug has on cardiac and vascular adrenergic receptors?
- A patient (76 years old female) with compromised cardiac function was hospitalized due to significant volume overload and generalized edema. Chest X-ray confirmed pulmonary edema, ejection fraction was estimated - 20%. Patients’ heart rate and blood pressure have been monitored. Despite intravenous use of diuretics she still feels significant shortness of breath, her blood pressure is now 90/60 mm Hg. Blood tests confirmed that renal function is worsening. Blood creatinine – 2.0 mg/L, Na – 132 mEq/L; K – 3.9 mEq/L. Intravenous therapy with Dobutamine was initiated. Two months later this patient was readmitted for an episode for cardiac arrhythmia. She was diagnosed with atrial fibrillation. Amiodarone was initiated for her arrhythmia treatment. Questions: What group of cardiovascular drugs does Amiodarone belong to? What is the mechanism of action? What effect this drug has on patient’s heart rate? What potential side effects can be related to long term Amiodarone use?шеa neat TosS evaporation is compromised first. OTrue False QUESTION 13 With each beat, the heart pumps: d. blood rich in oxygen to the lungs and blood rich in carbon dioxide to the body. b. OD. blood rich in carbon dioxide to the lungs and blood rich in oxygen to the body. C. blood rich in carbon dioxide to the lungs and back to the heart. d. blood rich in oxygen to the lungs and then back to the heart. QUESTION 14 Click Save and Submit to save and submit Click Saue All Ancugno to nauEdema would likely occur when blood hydrostatic pressure at the capillary decreases hemorrhage occurs blood volume decreases the concentration of protein in the blood increases O none of the choices would cause edema
- a man with hypertension takes a diuretic that is not potassium sparing and he does take potassium. What would be the side effect of this drug. 1. cardiac arrhythmia 2. depolarized neuronal cell membrane 3. increase blood volume 4. hyperkalemia 5. all of these would occurWhich of the following is a circulatory adjustment to sustained moderate exercise?a. Constriction of the cutaneous circulationb. Decreased intestinal blood flowc. Decreased venous returnd. Increased renal blood flowe. Increased total peripheral vascular resistance4. The heart, kidney, brain, retina and arterial blood vessels are prime targets of hypertensive damage. Uncontrolled and untreated AH accelerates the damage to these organs and could cause their failure.5. Medicines used to treat high blood pressure include: Water pills (diuretics), Angiotensin-converting enzyme (ACE) inhibitors, Angiotensin II receptor blockers (ARBs), Calcium channel blockers, Alpha-beta blockers,and etc6. Aldosterone (ALD) is a hormone your adrenal glands release that helps regulate blood pressure by managing the levels of sodium and potassium in your blood.
- Figure 1 shows the capillary exchange in the human blood circulatory system. Rajah 1 menunjukkan pertukaran kapilari dalam sistem peredaran darah manusia. Reabsorption Penyerapan H20 Filtration Penurasan Molecule B Molecule A Molekul A Co2 wastes semula Molekul B CO, Bahan buangan H,0 Capillary Кapilari Arterial end Hujung arteri Venous end Hujung vena Figure 1 Rajah 1 Name ONE example of molecule A. Namakan SATU contoh molekul A. Describe the transportation process of molecule B during capillary exchange. Terangkan proses pengangkutan molekul B semasa pertukaran kapilari. What will happen to molecule A if there was a plaque in the venous end? Apakah yang akan berlaku pada molekul A sekiranya terdapat plak pada hujung vena?Kidney failure would lead to the loss of the production of erythropoietin which untreated would lead to OA. Ischemic hypoxia OB. Anemic hypoxia OC. Hypoxic hypoxia D. Histotoxic hypoxia O E. None of the above1- explain why an increase in plasma viscosity will increase the ESR. 2- explain why spherocytes and sickle cells will decrease the ESR.
