why Penicillin inhibits PBP activity in wild-type cells

You are growing E. coli in a laboratory in order to study their operons. The growth media you are using contains lactose, no glucose and no tryptophan.  Using your knowledge of operons and their regulation, answer the following questions  I really need a in depth epxplantion for the queshtions to make sure i understand the answers    a.What effect does lactose have on the bacterial cell’s lac operon?    b.What does the absence of glucose do to the bacterial cell?    c.You now add tryptophan to the cell. What would happen to the bacterial cell and its trp operon?   d.What kind of regulation does lactose provide to an operon’s repressor?

How does tryptophan affect the function of trp repressor?

Repressor protein controlling own regulation in Lamda lysogenic cycle, justify with the following presentation.

The enzyme adenylyl cyclase is active only when glucose levels are high so that the lac operon can be turned on. True False

What is meant by the differential activation of genes? Explain how this affects the synthesis of cutin and P-protein.

explain the most likely steps of what might happen inside a MDA-MB-231 cell after it is exposed to UV light.

help

Which of the following mutations from question 2 could be restored to wildtype by adding CAMP as a supplement? O cya- where glucose cannot bind adenylate cyclase O cap- where CAMP cannot bind to CAP O cap- where CAP cannot bind to the CAP binding site O Mutation in the CAP binding site that prevents CAP from binding O cya- where adeynlate cyclase cannot turn ATP into CAMP

Give examples of nonsense-mediated decay (NMD) as a friend/foe. With explanation please. thanks