Why does neuronal function require the voltage-gated K+ channels to open more slowly than the voltage-gated Na+ channels and how would you expect the voltage-gated K+ channels to affect the shape of an action potential curve?
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Why does neuronal function require the voltage-gated K+ channels to open more
slowly than the voltage-gated Na+ channels and how would you expect the voltage-gated K+ channels to affect the shape of an action potential curve?
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- Assume presynaptic excitatory neuron A terminates on a postsynaptic cell near the axon hillock and presynaptic excitatory neuron B terminates on the same postsynaptic cell on a dendrite located on the side of the cell body opposite the axon hillock. Explain why rapid firing of presynaptic neuron A could bring the postsynaptic neuron to threshold through temporal summation, thus initiating an action potential, whereas firing of presynaptic neuron B at the same frequency and the same magnitude of EPSPs may not bring the postsynaptic neuron to threshold.What are THREE major functional characteristics of the voltage-gate Na+-channels and how do these characteristics contribute to the generation of action potential?If the response time of activation gates of both voltage gated sodium and potassium channels are same once induced by changes in membrane potential, what do you think will happen to generation of action potential when a strong (25 mV) graded potential reaches to axon hillock? Justify your answer with specific reasoning. thanks a lot in advance:)
- In an experiment, the extracellular [Na+] surrounding a nerve cell was reduced from 145 to 45 mM. Which of the following is the most likely effect of this on action potentials? No action potentials would occur because the concentration of extracellular Na+ is too low. The membrane potential would become more negative so the threshold for action potential generation could not be reached. The nerve cell would still produce an action potential but its amplitude would be reduced and the depolarisation phase would be slower. The nerve cell would still produce an action potential but its amplitude would be reduced and the depolarization phase would be more rapid.Which is TRUE about neuronal membrane electrical and concentration gradients at the peak of the action potential? The electrical gradient is in a direction that would tend to move K+ out of the cell. The concentration gradient for K+ is in a direction that would tend to move it into the cell. O The concentration gradient for K+ greatly increases compared to at rest. The concentration gradient for Na+ is in a direction that would tend to move it out of the cell. O The electrical gradient for Na+ is in a direction that would tend to move it into the cell.If action potential propagation from one neuron to another neuron requires ACh release, what event at the synaptic cleft explains the need for action potentials to arrive at a high rate before the post-synaptic membrane achieves threshold? the lack of sodium voltage gated channels on the post-synaptic membrane O the rapid breakdown of ACh by ACHE at the synaptic cleft the slow release of ACh by the synaptic vessicles the slow movement of sodium through chemical gates Previous Next MacBook Air
- Help me pleaseYou generate action potentials in a neuron bathed in solution in a petri dish by applying a threshold-level depolarizing stimulus near its axon hillock. If the solution surrounding the neuron contained 5mM K+, 150 mM Na+, and 0 mM Ca2+ which of the following would you expect? The neuron would not be able to propagate action potentials down the entire length of the axon The neuron would not release neurotransmitter from the axon terminal The neurons action potentials would have an unusually long duration The neuron would have a resting membrane potential of zeroName the three phases of an action potential. Describe for each the underlying molecular basis and the ion involved. Why is the term voltage-gated channel applied to Na+ channels involved in the generation of an action potential?
- If the response time of activation gates of both voltage gated sodium and potassium channels are same once induced by changes in membrane potential, what do you think will happen to generation of action potential when a strong (25 mV) graded potential reaches to axon hillock? Justify your answer with specific reasoning.The neuron resting potential diagram is confusing to me. For the supposed sodium potassium channel in purple, how come the sodium ions are going outside of the cell instead of inside? How is it that there is a higher potassium concentration inside of the cell at this time if there are more leaky potassium channels (which should be pumping the potassium ions outside)?What would occur if voltage - regulated Na+ and k+ gates opened at the same time rather than at different times, during the production of an action potential?