What is the PAO2 in our alveolar cells given that conditions are normal (room temperature) and at sea level, given that the the normal atmospheric pressure at sea level is 760 mmHg. The partial pressure for water is 47 mmHg and the PACO2 is 40 mmHg. The respiratory quotient is 0.8.

Compared to atmospheric air, the partial pressure of oxygen in the alveoli is (higher or lower) And the partial pressure of Carbon Dioxide in the alveoli is higher or lower?

How will changes in arterial Po2 impact ventilation? Select the one most accurate response. Small changes in arterial Po2 will result in fine changes to VÊ in order to maintain relatively stable arterial Po2. O Arterial Po2 will not affect ventilation unless it drops below 60mmHg. O₂ cannot cross the blood/brain barrier, thus cannot interact with central chemoreceptors to have an impact on ventilation. O Breathing 100% O2 at atmospheric pressure will greatly reduce the urge to breathe.

A spontaneously breathing patient has a normal PaCO2 of 40 mm Hg, a minute ventilation (VE) of 6 L/min, and a VD/VT ratio of 35% (0.35). Then the patient develops a pulmonary embolus (PE), blocking blood flow to a significant number of ventilated avleoli. Consequently, the measured VD/VT increases to 65% (0.65). 1. Calculate the alveolar ventilation associated with teh original PaCO2 of 40 mm Hg. 2. To maintain the same PaCO2 of 40 after the embolus occurs, what change must occur in total minute ventilation (VE)?

Does carbon dioxide move from the blood to the tissue cells or from the tissue cells to the blood and Why does it move in this direction (use it's partial pressures)?