If you were to apply a TRPM5 agonist to your tongue, which tastes would you likely sense?

Please help with complete answers of both the parts

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Briefly describe why there could be a possible link between TAS2R38 sensitivity and obesity. In your own words, formulate a summary (background, aim, method, results, conclusion) of a published study on obesity and taste receptors in your answer.

Please ASAP Thank you.  Give a breief explantion on neuro related disease Alzheimer? How it effecets a human life and the causes and consequences of it?

Superfamily Give the superfamily to which the receptor belongs Accession Give the Uniprot accession number Name Give the molecule name Species Give the species Ligand What is the ligand, or class of ligands which bind to this receptor? What are the physiological processes involved? Is this autocrine, paracrine or endocrine or some combination of them? What is the Key physiological pathology of the receptor? process involved What are the downstream actions of the receptor? Which molecules does it interact with, how, and to what effect? How rapidly doe the effects appear? Mechanism of action Give one key journal reference in Vancouver format that describes the mode of action of the receptor Key reference Focus dom)

Please ASAP. Thank you.

Dear Expert. Thank you for your advice, which is good and helpful. I note that your answer is "when 3 binding sites have been occupied the activity of these receptors is maximum." However, I also note that for a similar question (see below) on α7 nicotinic Acetylcholine Receptor (nAChR) also gives the same answer, ie. at least 3 agonist binding sites should be occupied.  Another Question - "To maximize the activity of an α7 nAChR, how many agonist binding sites should be occupied." I understand that in α7 nAChR, binding to two sites is more effective and binding to three sites is most effective for activation. On the other hand, binding to four or five sites in the nAChRs of chromosome 7 speeds up desensitization more than activation. Therefore, to maximize the activity of an α7 nicotinic Acetylcholine Receptor (nAChR) is also "3 sites". Am I right?

Can Please Help Me: Bio- Chemistry 60 minutes only the given time. Wish you could help me. I will give UPVOTE and GOOD FEEDBACK. QUESTION/// Diisopropyl fluorophosphate (DFP) inhibit acetylcholinesterase activity by reacting covalently to a serine residue found within the active site of the enzyme. The physiological effect of this inactivation is interference with neurotransmitter inactivation at the synapses of nerves, resulting in the constant propagation of nerve impulses, which can lead to death. What type of inhibitor is DFP? Explain. ///QUESTION

Please ASAP. Thank you. What types of GPCRs act in nociceptors to induce excitatory signaling?