Identify effects of chemicals (e.g. alcohol, caffeine, heroin) on synaptic transmission

Explain the importance of neurotransmitter removal from the synaptic cleft and describe three removal mechanisms.

Synaptogenesis is involved in long term memory consolidation. Describe the ways new synapses could form.

Identify the multiple mechanisms by which a drug can be pro-ergic or anti-ergic at a synapse.

READ: They key to large-scale communication in the entire nervous system is the ability to pass nerve impulses on a small scale from one neuron to the next so that signals can be transmitted throughout the entire body. The junction between two neurons is called a synapse, and is the critical space that mediates the transfer of a signal from the presynaptic neuron (the sender) to the postsynaptic neuron (the receiver) or even an effector cell (like a gland or muscle cell.) You circled a synapse in Model 1. Synapses can be electrical or chemical. Electrical synapses transmit electrical signals via gap junctions. A channel protein connects the cytoplasm of two adjacent neurons so that ions can flow directly from one cell to another to pass on the signal. However, the most common type of synapse is a chemical synapse. These utilize chemical signals, called neurotransmitters, to transmit the signal across the synaptic cleft. This is a slower process, but allows for much greater precision…

Short-term memory, like remembering a phone number, is stored as a Long Term Potentiation (LTP) of the synapse. Describe what has changed at the synapse when it has undergone LTP.

Define synaptic cleft.

Explain the propagation a nerve impulse and synaptic transmissions

Can you help me the name of the labels of the axon?

Discuss the mechanisms on how to stop the signal in synaptic transmission.