Phlebotomy Essentials
6th Edition
ISBN: 9781451194524
Author: Ruth McCall, Cathee M. Tankersley MT(ASCP)
Publisher: JONES+BARTLETT PUBLISHERS, INC.
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How would Cox-2 Inhibitors prevent the development of a peptic ulcer?
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- Exah Section Section 1: Mem 98 of 145 Mark 98. A 73-year-old woman has an adenocarcinoma within the left colic flexure that has penetrated the superior wal of the colon. Without restment, which of the bigges be invaded next by direct extension of the mass? A) Ascending duodenum B) Liver C) Pancreatic head D) Pyloric antrum National Board of Medica Gross Anatomy and Embryology OE) Spleenarrow_forwardIn regards to pancreatic cancer, are there any genetic mutations that lead to the condition?arrow_forwardage 4 a) pepsin trypsin breaks down dietary protelns and also activates the zymogen forms of other digestive proteases b) c) etrypsinogen, chymotrypsinogen, the pancreatic zymogens are procarboxypeptidase, and proelastase; the gastric zymogen is pepsinogen d) the mature, proteolytically active form of chymotrypsin consists of three peptide chains, A, B, and C, thát are held together by disulfide bonds the activation of chymotrypsin involves the conversion of chymotrypsinogen to e) the intermediate form a-chymotrypsin, which is then converted to the mature, fully active protease B-chymotrypsinarrow_forward
- 1: Bom 32 of 145 32. A 40-year-old woman with a history of gallbladder disease has pancreatitis. The most likely cause of the pancreatitis is a gallstone at which of the following sites? A) Common bile duct B) C) Cystic duct OD) Major duodenal papilla Common hepatic ductarrow_forwardNonsteroidal anti-inflammatory drugs inhibit the synthesis of prostaglandins, and can produce peptic ulcers. What function is normally ascribed to endogenous prostaglandins in the gut? Why prostaglandin synthesis inhibition can lead to the development of gastric ulcers? What preventive measures can be used?arrow_forwardHypothetically speaking, what would this graph suggest about the cause of ulcers? Assume Group 1 is antacid treatment and Group 2 is a placebo. # Ulcers/ patient Group 1 Treatment Group a) Antibiotics prevent ulcers b) Bacteria cause excess acid Group 2 Oc) The excess acid theory is correct d) The excess acid theory is incorrectarrow_forward
- A client with stomach cancer will receive nutrition via a tube that bypasses the stomach and goes directly into the jejunum. The nurse recognizes that the diet will need to be changed in which of the following ways? Question 28 options: a) Dietary protein will need to come from plant sources due to limited absorption b) The feed will contain di- and tri-peptides rather than whole proteins c) Proteins will require emulsification d) Conditionally essential amino acids will need to be added to the feedarrow_forwardDirect contact between intestinal bacteria and the epithelial cell surface is minimized by ?arrow_forwardZollinger-Ellison syndrome is a peptic ulcer disease characterized by overproduction of gastric acid. Which two types of stomach cells are most likely involved in Zollinger-Ellison Syndrome?arrow_forward
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