Flaccid paralysis (resulting from the failure of muscles to contract), induced by the neurotoxin tubocurarine, occurs when this poison blocks the binding of: the inhibitory neurotransmitter melatonin to post-synaptic bicarbonate channels the excitatory neurotransmitter glutamate to post-synaptic calcium channels the excitatory neurotransmitter acetylcholine to post-synaptic sodium channels the inhibitory neurotransmitter glycine to post-synaptic chloride channels the excitatory neurotransmitter serotonin to post-synaptic potassium channels

Dopamine is a neurotransmitter produced by the nervous system that is involved in the pleasure/reward system in the brain. The release of dopamine in response to a stimulus produces feelings of happiness and satisfaction. Cocaine is a type of drug that affects the brain response to dopamine. Usually after dopamine is released into the synaptic cleft, it is quickly re-absorbed into the neuron using dopamine transporters. However, cocaine blocks these transporters which causes dopamine to remain in the synaptic cleft for a long period of time. How would this impact the neuron's ability to communicate? Neurons will produce smaller action potentials because more dopamine is binding to receptors on the neuron, preventing the neuron from reaching the threshold potential Neurons will be under-stimulated because dopamine will bind to receptors and prevent ions from moving into the neuron, preventing electrical signaling Neurons will produce larger action potentials because more dopamine is…

In chemical synapses, neurons communicate with other neurons or skeletal muscles via neurotransmitters (NT). Explain the steps of neurotransmission beginning from the action potential arriving at the axon terminal to the termination of NT actions. (answer max 8 sentences)

Transcranial Magnetic Stimulation causes an involuntary motor output in response to a magnetic stimulation over the skull. Why do we see this output?  Include the following concepts in your response: •neuroanatomy •central versus peripheral nervous system •neuron structure and direction of information flow •hyper versus depolarization •specific ions •membrane potential •diffusion and electrostatic pressure