i need the answer quickly Explain the following effects of mineralocorticoids and glucocorticoidsa. What is the effect of aldosterone on sodium, potassium and water reabsorption in the kidneys?a. Why do different natural and synthetic glucocorticoids have different effects on sodium, potassiumand water reabsorption? What is the underlying mechanism behind these differences?NEED DETAIL EXPLANATION of both part lAS U CAN BUT IF U COPIED FROM INTERNET OR CHEGGSURELY GIVESU DOWNVOTEDon't give wrong answer otherwise downvote

a. Aldosterone acts on renal tubules to cause an increase in reabsorption of sodium ions and water…

Answered: a. What is the effect of aldosterone on…

Essentials of Pharmacology for Health Professions

7th Edition

ISBN:9781305441620

Author:WOODROW

Publisher:WOODROW

Chapter24: Reproductive System Drug

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Sorbitol accumulates in diabetics leading to reduced Na+/K+-ATPase activity in the retinal cells. Under these conditions, sodium levels inside the cells uptake, leading to leading to in glucose in collagen density, which may lead to blindness. O decreases, decreases, increases O decreases, increases, decreases O increases, decreases, decreases O increases, increases, decreases D increases, increases, increases E decreases, increases, increases KOIncreases, decreases, increases
2.2. Empagliflozin is an anti-diabetic medication used to improve blood sugar control in people with type 2 diabetes. Empagliflozin works by inhibiting the sodium- glucose co-transporter-2 (SGLT-2) found in the proximal tubules in the kidneys. Through SGLT2 inhibition, empagliflozin reduces renal reabsorption of glucose and increases urinary excretion of glucose. 2.2.2. Describe how the sodium-glucose co-transporter achieves its function at the molecular level. 2.2.3. Evaluate how the inhibition of SGLT-2 by Empagliflozin will affect the activity of the sodium-potassium pump. 3.1. Under normal physiological conditions, proto-oncogenes and tumour suppressors help control cell growth and proliferation. When mutated, these proteins contribute to the development of cancer. Detail the molecular basis of this phenomenon.
Ist this statement correct? Canagliflozin is therapeutically useful for managing type 2 diabetes because this drug decreases the renal clearance of glucose.
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op of Form Normal Levels of Substances in the Arterial Blood: pH 7.40 + 0.05 pCO2 (partial pressure of carbon dioxide) 40 mm Hg pO2 (partial pressure of oxygen) 90 - 100 mm Hg Hemoglobin - O2 saturation 94 - 100 % [HCO3-] 24 meq / liter Vignette #1: A 21-year-old noncompliant female with a history of type I (insulin-dependent) diabetes mellitus was found in a coma. Her blood glucose was high, as well as her urine glucose, urine ketones, and serum ketones. Her serum bicarbonate was < 12 mEq/L. Her respiration was exaggerated, and her breath had an acetone odor. Her blood pressure was 90/60 and his pulse weak and rapid (120). 1. Define noncompliant. 2. Is this person experiencing ketoacidosis or insulin shock? Explain your answer. 3. Why is the serum bicarbonate low? 4. What is the acid-base status of this individual? 5. What are the causes of the dyspnea, hypotension, and tachycardia? 6. What type of treatment does this person need?…
Understand Main Ideas 1. MAINIDEA Assess the reasons why hormone feedback systems are referred to as "negative feedback." 2. Predict when you would expect to find high levels of insulin in a person's blood and when you would expect to find high levels of glucagon in a per- son's blood.
thereof. 10. If you were working in a lab that studied IR and type II diabetes and needed to develop a drug to help treat type II diabetes, what would you want your drug to do and where would it be useful for it to interact? Explain why a drug with this mechanism of action would be helpful. Page "From Twiggy to Tubby" by Wilson, Rhodes, & Rozell
Select all of the following statentents that are a PLAUSIBLE explanation for the data Patient 1 (red) Patient 2 (blue) 10 200- 150- 150- 125- 100- 60 75 90 30 Time (min) 30 45 60 75 90 45 Time (min) O Patient 1 is insulin-deficient O Patient 1 is taking an SGLT2 inhibitor O Patient 2 is taking an SGLT2 inhbitor O The renal SGLT2 transporters saturate at lower threshold in Patient 2 than in Patient 1 The renal SGLT2 transporters have decreased function in Patient 1 Blood Glucose Urine Glucose
3:11 *il Xl 0.00 KB/S @ 34 ACTIVITY 4.do.. ACTIVITY 4- GLANDS Illustrate the MICROSCOPIC view of the following: I. Unicellular Glands 1. Goblet Cell Give one organ where a goblet cell is located b. Label the goblet cell а. II. Multicellular Glands A. Shape of Secretory Units 1. Tubular Gland 2. Acinar Gland 3. Tubuloacinar Gland Do these for each item: a. Differentiate each gland in one sentence only. B. Method by which the secretory product is discharged 1. Merocrine Gland 2. Holocrine Gland 3. Apocrine Gland 4. Endocrine Gland Do these for each item: a. Identify organs where you can find each gland. b. In one sentence or phrase, differentiate each gland.
In a different instance of two infants suspected to have a defi- ciency of the PDH complex because of severe lactic acidosis, tissue biop- sies could not be taken to measure URINARY EXCRETION OF PYRUVIC AND a-KETOGLUTARIC ACIDS Pyruvic Acid (итol/ mg of creatinine) a-Ketoglutaric Acid (µmol/mg of creatinine) Age Special Diet or Drugs Patient G. M. 5 wk 2.26 3.57 enzyme activities. Instead it was no- ticed that there were high concentra- tions of organic acids in the urine. Typical results are tabulated in the ta- ble on the right. Identify the enzyme defect and explain why it presents this metabolic pattern. What other meta- bolic breakdown products likely were also detected especially following a high protein diet but are not listed? Name the defective enzyme. 7½ wk 2.10 3.39 11 wk 1.35 4.63 12 wk After 18-hr fast 1.87 0.44 High-protein, low-carbohy- drate diet 15 wk 1.62 5.38 Patient D. M. 1 day 13 days 0.87 0.44 Thiamine 0.45 3.44 24 days 0.33 2.47 4 wk 0.53 1.23 6 wk Thiamine 0.42…
B. Blood Pressure Homeostasis Use the terms in bulleted list to complete the schematic diagram of blood pressure homeostasis in Figure 29.8. ram of blood p • ADH • aldosterone blood pressure G 1 HOMEOSTASIS asmul sit widiw bootd cili mon ainorouh Normal BP and volume to low ons nidhiwandi steun dioome sifT.C Stimulation Short Term Regulation Nervous System Stimulation of baroreceptors and chemoreceptors Stimulation of cardiovascular center 3 stimulation e-Leaf Print Companio HYSIOL Decreased BP and volume Peripheral vasoconstriction blood volume • cardiac output sympathetic nerves 5 FIGURE 29.8 Blood pressure homeostasis. Renin (kidney) Angiotensin II activation Homeostasis restored Long Term Regulation Hormones 6 analligas om nidhiw well boolll L 4 (Posterior pituitary) (Adrenal gland) 2018 A mood: How alov Blood pressure ΕΡΟ erythropoietin (kidney) RBC production
A physician orders lidocaine 100 mg in D5 How many drops per minute will be administered? sgad to ini yasm voH A physician orders epinephrine 50 mcg/min to be administered from fluids that contain epinephrine 5 mg in D5LR 500 mL. The drop factor is 20 gtt/mL. How many minutes will it take for the fluids to infuse? What is the rate of infusion in drops per minute? What is the flow rate in milliliters per hour? after 300 mL had infused? Luhte reserved.

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a. What is the effect of aldosterone on sodium, potassium and water reabsorption in the kidneys? a. Why do different natural and synthetic glucocorticoids have different effects on sodium, potassium and water reabsorption? What is the underlying mechanism behind these differences?