Between your evening meal and breakfast, your blood glucose drops and your liver becomes a net producer rather than a consumer of glucose. Which statements describe the hormone-stimulated process that triggers glucose production by the liver? The increase in (CAMP] causes protein kinase A to phosphorylate pyruvate kinase, thus inhibiting glycolysis in muscle tissue to promote gluconeogenesis in the liver. O The decrease in [fructose 2,6-bisphosphate] stimulates FBPase-1, the key enzyme in gluconeogenesis. Epinephrine stimulates an enzyme cascade involving hepatic protein kinase A (PKA) and phosphorylase b kinase, which eventually leads to glycogen breakdown in the liver. The rapid decrease in blood glucose levels triggers glucagon release by the pancreas. O Glucagon stimulates CAMP-dependent phosphorylation of PFK-2/FBPase-2, which decreases [fructose 2,6-bisphosphate). The decrease in [fructose 2,6-bisphosphate] stimulates PFK-1, the key enzyme in gluconeogenesis.

The main enzyme of glycogen degradation is [Select] is low, this enzyme is activated by [Select] [Select] When blood glucose in response to the hormone

vny can acetone sometimes be detected in the breath of patients with type 1 diabetes? A. Glucagon signaling is faulty, leading to an excess of oxaloacetate, which enables acetyl-CoA to enter the citric acid cycle, therefore resulting in the production of ketone bodies including acetone OB. Insulin signaling is faulty, so excess malonyl-CoA is generated, which leads to excess acetyl-CoA from beta oxidation Oc. None of the other answers is correct OD. Insulin signaling is correct, but excess acetyl-CoA is generated anyway, leading to the production of ketone bodies including acetone O E. Insulin signaling is faulty, fatty acids are catabolized to acetyl- COA despite high blood glucose, and excess acetyl-CoA is converted to ketone bodies,

Gluconeogenesis occurs in liver and kidney. Which is of the following enzyme are important for gluconeogenesis are expressed exclusively in these tissues? Select one: a. Glucose-6-phosphatase b. Fructose-1,6-Bisphosphatase c. Phosphoenolpyruvate carboxykinase d. Pyruvate carboxylase

Glucose can be made from oxaloacetate during gluconeogenesis, but if oxaloacetate concentrations are decreased,what other substance can be used to make glucose? How might this contribute to increased fat loss?

Which of the following glycolytic enzymes catalyzes the conversion of fructose-1,6-bisphosphate into the two product molecules dihydroxyacetone phosphate, and glyceraldehyde-3-phosphate? phosphoglycerate kinase pyruvate kinase malate dehydrogenase aldolase phosphofructokinase

Which of the following statements is true for the shown reactions?   Deficiency of oxaloacetate stimulates the formation of X and y Insulin signaling stimulates the formation of X and Y Both A and B Neither A nor B

Glucagon  The function of glucagon is in promoting energy release and inhibiting energy storage  in response to low circulating glucose levels, which are largely controlled by insulin [24]. Besides its role in blood–glucose regulation, patients treated with glucagon receptor an  tagonists (GRA) often suffer from dyslipidemia, fatty liver and weight gain, suggesting  that glucagon play an important role in lipid metabolism [25]. Glucagon acts mainly on  the hepatocytes which possess the highest level of glucagon receptors. Following mecha nisms may be involved during this process: (1) At the molecular level, following glucagon  binding to its receptor on the hepatocyte, cAMP will be activated and accumulated, which  in turn activates cAMP-response element-binding protein (CREB). As a result, the tran scription of carnitine acyl transferase (CPT-1) increases, which converts fatty acids into  acylcarnitine and activates β-oxidation-increasing fatty acid catabolism [26]. In addition,…

Plase answer this question and the highlighted question is wrong , provide me a explanation why it is wrong and correct answer.

Which changes would be expected in a normal human after a meal rich in carbohydrates (select all that apply)? O increased secretion of the pancreatic hormone, insulin O increased glucose uptake in myocytes via glucose transporters increased activity of glucose 6-phosphatase in hepatocytes O increased activity of the liver enzyme, glucokinase O increased activity of GSK-3 in both myocytes and hepatocytes