You are studying Hox genes in crane flies (Leptotarsus testaceus). The cranefly genome is sequenced, and you have access to this sequence. You are interested in studying the EVE gene in craneflies. Using your understanding of Hox genes, design an experiment testing where the homolog of the EVE gene is expressed in cranefly embryos.

After graduating from UNC Charlotte with the BS in Biology, you get a job with an agro-chemical company and are assigned to a lab that is exploring the use of a newly synthesized compound that may be a possible insecticide. The compound is thought to control insect populations by disrupting the genes that control embryonic development. Your lab conducts an experiment to investigate the influence of the compound on developmental genes by measuring the levels of the proteins the genes code for.  You measure protein levels in two groups of insect eggs: the Treatment Group, which is exposed to the compound, and the Control Group, which is exposed to a compound that is known to have no negative effects on gene activity. Your results are shown below. In all graphs, the Control group exhibits normal levels of proteins and gene expression. (Remeber: P > 0.05 means the observed differences are not significant; P < 0.05 means the differences are significant and biologically meaningful).…

Why did the mouse Pax6 gene produce fruit fly eyes instead of mouse eyes?

Explain about how X chromosome inactivation takes place in mammals at the chromosomal and molecular level.    B. What genes are involved in inactivating a human X chromosome?  . C. What role does  X chromosome-specific inactivating transcript --XIST-- non coding RNA play in the inactivation of the human X chromosome.

The functions of almost all of the genes in the lambda genome were first explored using mutations. What do you think the mutant phenotype would be if you made an inactivating mutation in the c gene, and why? Be very specific but also very brief.

Please answer

Why is an understanding of gene regulation in eukaryotes crucial to an understanding of developmental processes? Explain your answer

And see the following question correctly thanks

In the introduction to this chapter, we discussed the role of famine during early prenatal development and how, through epigenetic effects, it influenced the health of individuals as adults. One effect that has been observed in people who were conceived during the Dutch Hunger Winter is that they suffer more cognitive decline later in life. If these individuals were autopsied after death, what differences might you expect to see in the chromatin of their brain cells? What do the mouse studies of cognition and epigenetic effects suggest?