Initial Response to Exercise

You may list, as students report out, the physiological changes to the respiratory, cardiovascular, neuromuscular, and urinary systems expected during strenuous exercise and as noted in the case of the cyclist, Joe. Students will respond with answers suggesting increases in heart rate, respiration, sweating and muscle fatigue, as well as muscle soreness as normal. However, in

As a result of the contractions in the Muscle- Skeletal Longitudinal Section cells and the Muscle- Skeletal Cross Section cells, it allows your muscle to be able to contract in response to nerve stimuli. This means that the movements of most of these muscles are not involuntary, you can control them. Therefore, once the stimulation stops, the muscles relax.

P6- follows guidelines to interpret collected data for heart rate, breathing rate and temperature before and after a standard period of exercise

In this assignment I will be reviewing the different effects of exercise on the body system including the acute and long term using the pre-exercise, exercise and post-exercise physiological data which I collected based on interval and continuous training method. I will also be including the advantages and disadvantages of these, also the participants’ strengths and areas where they can improve on.

Brooks GA, Fahey TD, Baldwin KM (2005). Exercise Physiology: Human Bioenergetics and Its Application. 4th Edition

Exercise increases heart rate by a process of sympathetic autonomic stimulation. Sympathetic (adrenergic) nerves increase the excitability of the sino-atrial node and reduce the P-R interval .As exercise continues, the physiological changes in the body are continuously monitored by a number of physiological systems and the balance of activity of the sympathetic system (speeding up) and the parasympathetic system (slowing down) is constantly adjusted. When exercise is over, the heart rate does not drop immediately as the body has to undergo a period of re adaption to return to the resting state.

D1 outline the relationships between the cardiovascular, respiratory and energy systems before, during and after a sporting activity

During exercise there is an increase in cardiac output, which corresponds to an increase in maximal oxygen consumption. With the increase in oxygen consumption, a greater increase in blood flow occurs. This means there is more oxygen circulating in the blood for the tissues to take up. Due to the increase in blood flow, vasoconstriction of arterioles occurs to maintain mean arterial pressure (Bassett & Edward, 1997). This limits oxygen consumption because some of the blood flow is directed to the brain and skin. It is further pointed out that the heart is another limiting factor because it determines how much blood and oxygen are supplied to the muscles especially when blood flow exceeds maximal cardiac output (Bassett & Edward,

Muscle contraction can be understood as the consequence of a process of transmission of action potentials from one neuron to another. A chemical called acetylcholine is the neurotransmitter released from the presynaptic neuron. As the postsynaptic cells on the muscle cell membrane receive the acetylcholine, the channels for the cations sodium and potassium are opened. These cations produce a net depolarization of the cell membrane and this electrical signal travels along the muscle fibers. Through the movement of calcium ions, the muscle action potential is taken into actual muscle contraction with the interaction of two types of proteins, actin and myosin.

trained athletes will have a lower heart rate during this period of exercise. Recovery heart rates –

Heart rate anticipatory response – this is where the heart rate starts to automatically increase before you start to exercise. The heart rate is able to increase automatically by chemical hormones, the hormones are adrenaline and noradrenaline. These hormones are found inside the brain. The reason the heart rate increase before exercise is because it prepares the muscles for exercise, the reason it prepares the muscles for exercise is because by the heart rate increase the more oxygen is getting to the muscles there fore they will not be needing a such a large oxygen supply all at once. It doesn’t only supply oxygen it supply’s nutrients, the supply of nutrients also provides energy and helps to repair the muscles after exercise. By the heart rate starting to increase gives the heart a head to start pumping hard this enables the heart to not have as much stress on it.

To maintain effectiveness of muscle and bone activity, the effects of on the musculoskeletal system are the greatest benefits a person can ask for.

As the intensity of exercise increased, so did the rates of the heart and breathing. After a small period of rest, the heart rate and breathing rate both decreased to a point close to their resting rate. This proved the stated hypothesis. First, the hearts average resting rate was recorded to be 76 bpm. The heart is therefore transporting oxygen and removing carbon dioxide at a reasonably steady rate via the blood. During the low intensity exercise (Slow 20) the heart rate increases to 107 bpm, which further increases to 130bpm at a higher intensity level (Fast 20). The heart therefore needs to beat faster to increase the speed at which oxygen is carried to the cells and the rate at which carbon dioxide is taken away by the blood.

Aims - This study is to ascertain, if there is an effect on heart rate after exercise. This is being done to see, if there is a difference between resting heart rate and heart rate after performing exercise.

The literature on the effects of exercise of cardiac output maintains the idea that exercise should affect cardiac output- pulse rate, systolic blood pressure, diastolic blood pressure, QRS-pulse lag, P-T and T-P intervals, because of increased heart rate. For our experiment, we tested this theory by measuring our cardiac output before and after some rigorous exercise. We measured the individual cardiac output and then combined the data to compose a class-wide data average. We compared the results of the experiment to what we expected, which was that exercise does affect our heart. Our data from this experiment supported the notion that exercise does, in fact, change cardiac output.