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Antisocial Personality Disorder ( Aspd ) Exhibit

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As defined by the American Psychiatric Association (2013), individuals diagnosed with antisocial personality disorder (ASPD) exhibit “a pervasive pattern of disregard for, and violation of, the rights of others that begins in childhood or early adolescence and continues into adulthood. Individuals, both with and without ASPD, often find themselves facing stiff consequences when they break the law by violating the rights of others. The propensity of individuals with ASPD to break such laws is one possible explanation for the prevalence of ASPD increasing from a range of 0.2% to 3.3% in the general population to 47% in prisons (American Psychiatric Association, 2013; Fazel & Danesh, 2002). With such a disparity between the general and prison prevalence of ASPD, it is clear that society is adroit in isolating the undesired behaviors of individuals with ASPD; however, society also has a responsibility to protect the rights of those diagnosed with a bonafide illness. If ASPD is the cause of an individual’s incarceration, then their successful reintegration into society relies on treating the underlying diagnosis, and in order to do that, it is important to understand the neurobiological foundations of ASPD. Individuals with APSD exhibit neuroanatomical differences compared with individuals without the diagnosis. Magnetic resonance imaging (MRI) studies have identified an 11.0% reduction in prefrontal gray matter in individuals diagnosed with ASPD (Raine, Lencz, Bihrle, LaCasse, &

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