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- 2 3 inte tion of Lormhda. ne att si ucated betwee de ne att sites and go ns one te n vacter aged ot utilize doot ractose ato la t don s gal* uc Integration to form prophage om SOward cor coct some cro on! ni imple ex 2 3 bio* gal* 14. The figure provided portrays the integration of Lambda phage into a host chromosome at the att site, located between the gal+ and bio+ genes. This prophage may disintegrate from the host carrying with it host genes, such as gal+ and/or bio+ and go on to transduce another host bacterium. How would one determine if a gal- host bacterium's phenotype was changed from gal- to gal+? To clarify, the minus version cannot utilize galactose as a carbon source for growth because it does not produce galactase, the enzyme that hydrolyzes galactose into monomeric sugars. There is a straight forward answer/solution to this – do not concoct some crazy solution! Think simple experiment.Qy: d whicel folowing Sucturs reprasent Perfeet orl trap? why? One TheThe pireaple Ssynaling prthony is asa commbly dsagphad in some fiype of Cancers o growth factor Pineappte reltptor Coconut time for Lell. Divsion "OFE Juity ON" When it is time for the cell to Mivilde, other ello in the backy helkare gouth factor molecdes, These bind to the pineapple receptoss in And the cell menmbane a One ths mppens, the toP protein becomes aitire. TOP In turn activates iy protein Which drives cell cyde forwarel. Howere, Hs mportant that the cell not divide centinuadly. Coconut protcin makes Sure Suiy tuns off once the cell completes divisiona Questions ) Top prokin is a und a mutation could enuse Lancer. 2) Juiy proein i5 a und a mutution coreld cause Lancer : 3 Cocoaut protkcin is a aod a mitution ceeled lnse canor word bank LOF, GOF, tunur Proto -oncoprotoin suppressor,
- Gone X is responsible for cell poiteratian and when gene x is expressed, me cells divide e and go through Xhe henmau ceu cycie Wren is not present, proliferanan ends. a) what is oa dominant mutanon that can occur in have a dominant effect on celu proliferation? And wnere in genex waud me mutaion occur li.e.pomoter? ete) gene X gene x not uould b) what is a recessive mutation that can occur in ingene X that would have recessiue effect on prouperation and where in genex would the mutanon occur?Can we identify general featurescommon to all cancer cells—such astheir production of misfolded, mutatedproteins—that can be used for thetargeted destruction of many differenttypes of cancers?why wxactly 20Atp not 24?
- ocument/d/1J-wo90GpYsd_jQSUBtDQHWisqGvSOteUQYoXaXazyS0/edit uction to cell.. R 1 Summary of Philo... E Petrona Andres Mig.. 2 Translations IXL: Par... IXL - Translations: g.. 1 IXL- meostasis Lab Exercise Tools Add-ons Help Last edit was 2 days ago text Calibri 12 BIU Conclusion: 1. List the changes you observed in the body color and perspiration level in response to? 2. Explain how the changes help the body adjust to maintain equilibrium (homeostasis)? 3. Speculate why a change in body temperature occurs? 4. Name which mechanisms your body uses to maintain a constant body temperature? 5. Explain why an increased breathing rate accompanies exercise? 6. Explain why an increased heart rate accompanies exercise? 7. Write a paragraph about the conclusions you can draw about your body's ability to maintain equilibrium (homeostasis). Be sure to include the answers to the questions above.10- 00 O SCNSA-SCN10A CDKN1A HANDI VTI1A SYTI • MYOCD 9 10 11 12 13 14 15 16 17 18 19 20 2122 X YWhat is the type of geneticinheritance of daltonism? Isdaltonism more frequent inmen or in women? What is thephysiological explanation forthe daltonism?
- explain F' plasmid formationBasis of crown gall tumor development by Agrobacterium tumefacien?Before development of a vaccine against this microbe, thedisease it caused accounted for two-thirds of bacterial meningi-tis cases during the first year of life but is still the number oneleading cause of mental retardation in patients who survive seri-ous disease due to permanent central nervous system disorders.What is the microorganism?(a) Haemophilus influenzae type B(b) Haemophilus influenzae type A(c) Neisseria meningitidis(d) Streptococcus pneumoniae(e) Listeria monocytogenes