Illustrate a SARS-CoV-2 genome organization showing all the genes. Label the 4 structural genes and the 2 genes that code for the PP1a and PP1b and produces 16 nonstructural proteins.
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Illustrate a SARS-CoV-2 genome organization showing all the genes.
Label the 4 structural genes and the 2 genes that code for the PP1a and PP1b and produces 16 nonstructural proteins.
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- Using PEN and PAPER ONLY, illustrate a SARS-CoV-2 genome organization showing all the genes.Label the 4 structural genes and the 2 genes that code for the PP1a and PP1b and produces 16nonstructural proteins.describe SARS-CoV2 Spike Proteinut the following steps of SARS-CoV-2 replication in order ag and drop options into correct order and submit. For keyboard navigation... SHOW MORE ✓ ||| = ||| = ||| ||| ||| = Virus binds to a receptor on a human cell's membrane Virus releases its RNA genome into the cell New virus travels to the cell membrane of the host cell and is released outside the cell. Viral RNA genome is translated into proteins by the host cell's ribosomes Viral RNA dependent RNA polymerase helps transcribe more copies of the virus's RNA
- The SARS-CoV-2 genome is a polycistronic genome. What does the term polycistronic refer to3.) A number of publications explain an expanding knowledge on the genetics and genomics of SARS-CoV-2 and its implications for understanding COVID-19. Why should a healthcare professional need to have a solid understanding of these basic genetics and genetic engineering terms? How would this knowledge help you in your nursing practice? 4.) Scientists and researchers are using genetic engineering rather than traditional methods in developing COVID-19 vaccines. In a tabular format give at least 2 examples of vaccines that uses genetic technology and describe each in terms of: a. Target Population b. Vaccine Efficacy C. Vaccine administration d. Possible side effects Storage requirements e.A snapshot of a portion of the SARS-CoV-2 spike protein bound to the human ACE2 receptor (taken from PDB) is shown below. The spike protein is in orange and the ACE2 receptor is in green. Within the red-circled region are 2 amino acids connected by a dashed line (added by Dr. David). These amino acids are a tyrosine residue from the spike protein and an aspartate residue from the ACE2 receptor. w cozed
- The SARS-CoV-2 genome consists of positive sense mRNA. What does positive refer to?For the following diseases, describe the best technique for diagnosing them. Please make sure you include how you would tell someone with the disease from someone without the disease. B. Factor V Leiden thrombophilia is caused by a point mutation at position 1691 in exon 10 of the Factor V clotting factor gene that changes an arginine into a glutamine. This change removes one of the cleavage sites for activated protein C and leads to an increased tendency to clot.Even single point mutations can have a significant effect on protein function. Describe how point mutations in the SARS-CoV2 spike protein might affect its function.
- 1) Define each of the following abbreviations: COVID-19: 2019-nCoV: SARS-CoV-2: 2) How does SARS-CoV-2 differ from SARS-CoV? 3) What is meant by the terms "delta variant" and "omicron BA.2" variants? How many waves of global variant spread have we had to date? 4) What are the defining chemical features of the coronavirus? Is this an enveloped or non-enveloped virus? How does this impact our ability to neutralize the virus outside of the body? What type of nucleic acid does it contain? (RNA? DNA? Single-stranded? Double-stranded?) Are coronaviruses genetically stable or highly variable? How does this correlate with their genetic material?We looked at RNA-mediated mechanisms of gene regulation in both eukaryotes and prokaryotes. Discuss the similarities and differences. How can you use this information to design therapy for SARS-Cov-2.Discuss what kind of results do you expect to see with your choice of design.Make sure you have at minimum watched the WK11 Concepts in Context videos on Spinal Muscular Atrophy. Spinal Muscular Atrophy: “Mechanism of Splicing Regulation of Spinal Muscular Atrophy Genes” (2018) https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6026014/ Abstract: “Spinal muscular atrophy (SMA) is one of the major genetic disorders associated with infant mortality. More than 90% cases of SMA result from deletions or mutations of Survival Motor Neuron 1 (SMN1) gene. SMN2, a nearly identical copy of SMN1, does not compensate for the loss of SMN1 due to predominant skipping of exon 7. However, correction of SMN2 exon 7 splicing has proven to confer therapeutic benefits in SMA patients. The only approved drug for SMA is an antisense oligonucleotide (Spinraza™/Nusinersen), which corrects SMN2 exon 7 splicing by blocking intronic splicing silencer N1 (ISS-N1) located immediately downstream of exon 7. ISS-N1 is a complex regulatory element encompassing overlapping negative motifs and…