Flaccid paralysis (resulting from the failure of muscles to contract), induced by the neurotoxin tubocurarine, occurs when this poison blocks the binding of: the inhibitory neurotransmitter melatonin to post-synaptic bicarbonate channels the excitatory neurotransmitter glutamate to post-synaptic calcium channels the excitatory neurotransmitter acetylcholine to post-synaptic sodium channels the inhibitory neurotransmitter glycine to post-synaptic chloride channels the excitatory neurotransmitter serotonin to post-synaptic potassium channels

Flaccid paralysis (resulting from the failure of muscles to contract), induced by the neurotoxin tubocurarine, occurs when this poison blocks the binding of: the inhibitory neurotransmitter melatonin to post-synaptic bicarbonate channels the excitatory neurotransmitter glutamate to post-synaptic calcium channels the excitatory neurotransmitter acetylcholine to post-synaptic sodium channels the inhibitory neurotransmitter glycine to post-synaptic chloride channels the excitatory neurotransmitter serotonin to post-synaptic potassium channels

Human Physiology: From Cells to Systems (MindTap Course List)

9th Edition

ISBN:9781285866932

Author:Lauralee Sherwood

Publisher:Lauralee Sherwood

Chapter4: Principles Of Neural And Hormonal Communication

Section: Chapter Questions

Problem 13RE

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If a patient was experiencing difficulty controlling their movement and displaying motor tremors and this problem was seen ONLY in the central nervous system (not PNS), what system in the central nervous system is likely where the dysfunction is located, what neurotransmitter would likely be related to these symptoms, and what would be the effect of blocking reuptake for this neurotransmitter if the patient was given a reuptake inhibitor for this neurotransmitter?
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