Prostate

PSYCHOLOGICAL AND PSYCHOSEXUAL ADJUSTMENT OF PROSTATE CANCER Sexual dysfunction represents a prevalent long-term complication among prostate cancer survivors with a wide spectrum of manifestations and a huge impact on quality of life [4]. Resnick et al (2013) reported long-term effects of sexual function after radical prostate cancer treatment reported by prostate cancer survivors. As shown in a 15-yr follow-up study consisted of 1655 men who had localized prostate cancer the decline in erectile function

The prostate is a walnut sized shaped gland surrounding the top of the urethra whose growth and function is controlled by hormones such-as testosterone. A normal prostate in adult men has a mean weight of about 11g. The function of the prostate is to produce a majority of seminal fluid (Marker et al. 2003). Prostate cancer (PCa) is the commonest malignancy tumour in men and is second in cancer related death after lung cancer. PCa is mainly adenocarcinomas originating from the cortex of the gland

Prostate cancer is now the most common cancer in men in the UK and the second highest cause of male cancer death. Yet despite its high prevalence it is also highly survivable, with figures from Cancer Research UK showing that 84 per cent of men diagnosed with the disease survive ten years or more following treatment. With such a high survival rate would be easy to assume that the needs of this patient group are being adequately met. But this isn’t the case, says specialist prostate cancer physiotherapist

Prostate cancer is the one of the most lethal disease in the United Sates. The pattern of disease recurrence being the major cause of morbidity and mortality. In spite of recent advances in our understanding of the molecular mechanisms responsible for the development of prostate cancer, the survival rate of men with this disease has remained relatively unchanged in over the decades. Since the Nobel prize winning discovery by Dr. Huggins and Dr. Hodges (Huggins C., 1941) androgen deprivation therapy

A Literature Review of the Journal Club Article Entitled “Facilitating Shared Decision Making About Prostate Cancer Screening Among African American Men” According to the ACS Statistics of cancer incidence rates among US males for 2003-2007, prostate cancer occurs more commonly in African American men than in white males (ACS, 2011-2012). Therefore, it is not only essential, but also vital to increase knowledge on the subject matter, raise general awareness, and promote shared decision making about

They found that the solution lied in the saturation, where the maximal growth of the prostate cancer was achieved at a low level of testosterone. This model was produced by Fowler and Whitmore, who concluded “normal endogenous testosterone levels may be sufficient to cause near maximal stimulation of prostatic tumors.” There final conclusion was that “there is not today—nor has there ever been—a scientific basis for the contention that a higher T concentration causes pCA growth, acutely or long-term

In 1853, J. Adams, a surgeon at The London Hospital, documented the first case of prostate cancer. This was discovered by a histological examination. He documented in his report that this condition was “a very rare disease.” (Adams, 1853) At that time it was not clear to track the trend of prostate cancer as it now poses as a significant health problem. “In the United States, it is the most commonly diagnosed cancer in men, with 180,000 new cases and about 31,000 deaths occurring annually.” (Greenlee

What is the prostate gland and where it’s located The prostate gland is about the size of a whole-nut in most men, it consists of 70% glandular tissue and 30% muscular tissue [2]. [Figure 1] shows that the prostate sits at the bottom of the pelvis right under the bladder, the floor of the bladder is basically the top of the prostate (it adheres firmly to the base of the bladder). The rectum wraps around the back of the prostate so the front wall of the rectum is lying over the prostate itself. The

The finding led to the belief that the solution lied in the saturation, where the maximal growth of the prostate cancer was achieved at a low level of testosterone. This model was produced by Fowler and Whitmore, who concluded “normal endogenous testosterone levels may be sufficient to cause near maximal stimulation of prostatic tumors.” There final conclusion was that “there is not today—nor has there ever been—a scientific basis for the contention that a higher T concentration causes pCA growth

hard-to-cure disease like cancer. Prostate Cancer is a known cancer that happens to men only and a lot of men in the U.S are diagnosed with it, and happens to male’s reproductive system. Specifically speaking, it is a gland found in the most private area of a man. In the following paragraphs, I will be explaining why we need to find a way to combat prostate cancer, how to detect this cancer at early stages, and the risks associated with prostate cancer. Prostate cancer is the second leading cause