Would a chemical that interacts with Ftsl, preventing these proteins from functioning, make a good antibiotic? Why or why not?

How do populations (or strains) of microbes evolve resistance to antimicrobials? (For this question, explain how random mutations and the variation in susceptibility within a population affect the development of resistant strains?

Please explain how different detergents take out proteins selectively out of membrane. (Choose one protein as an example from the figure)

Based on your disc susceptibility results for E. coli R-, which ONE of the following is correct if considering treatment of an infection caused by this organism? Select one: A. All drugs evaluated could be considered for treatment in this case  B. Zone sizes were too large so the results are inconclusive and should be repeated C. Ciprofloxacin is the most potent drug so this should be used preferentially over all others D. Nitrofurantoin and fosfomycin are the most potent so either drug should be used preferentially over all others E. High drug potency is associated with increased toxicity so the drug with the lowest potency should be considered preferentially

nich TWO of the following statements about Extended Spectrum ß-lactamases are CORRECT? O Extended Spectrum ß-lactamases can inactivate all B-lactam antibiotics except third generation antibiotics in this family. O Genetic material encoding for extended spectrum ß-lactamases are easily transferred to other bacteria via transduction. O Extended Spectrum ß-lactamases are frequently produced in Klebsiella sp. and Escherichia coli strains. O Many Extended Spectrum ß-lactamases are not inhibited by clavulanic acid and other B-lactamase inhibitors. OSome Extended Spectrum ß-lactamases-producing E. coli are still sensitive to penicillins. OA new class of Extended Spectrum ß-lactamases has emerged and these have been widely detected among S. epidermidis. O Extended Spectrum ß-lactamases are not effective against the cephalosporin family of antibiotics.