Why is the rate of cardiac muscle relaxation faster when a positive inotropic agent is present?

Unlike skeletal muscle, cardiac muscle doesn’t undergo tetany. Explain how this is achieved and why is this necessary?

5) what causes the plateau phase during a cardiac muscle action potential? a) open voltage gated K+ channels b) closed voltage gated K+ channels c) open voltage gated Ca+ channels d) open voltage gated Na+ channels e) closed voltage gated Ca+ channels

What are the two functional importances of the intercalated discs of cardiac muscle?

In a cardiac contractile cell what is the mechanism involved in the change in membrane potential? Depolarization: P wave Repolarization: QRS Complex, Hyperpolarization: T wave Pacemaker: K+ and Na+ flow, Depolarization: Pca rises Repolarization: Pk rises Depolarization: Pna increases, Peak: Na channels close, Plateau: Calcium channels close & Potassium channels open, Repolarization Potassium flows out & Calcium flows in Pacemaker: K+ and Na+ flow, Depolarization: Pca rises Repolarization: Pk rises, 4. If channels close Depolarization: Pna increases, Peak: Na channels close, Plateau: Calcium channels open & Potassium channels close, Repolarization Potassium flows out & Calcium flows out

Describe the conditions at the sarcolemma of cardiac muscle cells at rest.

What action potential of specialized heart cells know the function of each channel and the process?

Frank-Starling law of the heart states: if you increase the stretch of the cardiac muscle fibers you will decrease the force of contraction a greater end diastolic volume will produce a weaker contraction of the ventricle by increasing the force of ventricular contraction you will increase the end systolic volume if you increase the stretch of the cardiac muscle fibers you will increase the force of contraction.

Which of the following statements best describes the differences in the regulation of cardiac and skeletal muscle contraction?     The amount of contractile force actively generated by muscle cells is increased by stretch in skeletal muscle and decreased by stretch in cardiac muscle.     Cardiac muscle is stimulated by motor neurons and skeletal muscle by neurones from the autonomic nervous system.     Skeletal muscle contractile force is augmented by increasing the firing frequency of action potentials whereas cardiac muscle contractile force is enhanced by noradrenaline increasing calcium influx through ion channels.     Ryanodine receptors in skeletal muscle are opened by a mechanism that requires calcium influx whereas in cardiac muscle membrane depolarisation alone without calcium influx is sufficient to open ryanodine receptors.

Why is afterload important to cardiac function?