Why is the lactic acid test done when sepsis is suspected?  Explain why the body produces excess lactic acid during sepsis (Be sure to cover all possibilities). How does this relate to fermentation and final electron acceptors during metabolism?

hyperammonemia in patients is compounded by the reality of the normal regulatory controls in the urea cycle. Explain how increased GDH oxidative deamination activity will inhibit the urea cycle.

How do alcohol and lactic acid fermentation differ other than their final product? (Hint: Look closely at the reduction reactions taking place in each pathway)

+ edu.au/courses/26618/quizzes/67364/take The image below shows the urea cycle. Based on the information in the image, which of the following is the most effective way of reducing the production of urea? CNH, Fumarate Arginine H₂O Arginase NH3+ C=N Arginino- succinate AMP + PP₁ NH₂ Arginino- succinase UREA CYCLE Urea ATP Argininosuccinate synthetase Ornithine H₂N H₂N 2 Citrulline Rate- limiting Ornithine 1. Carbamoyl phosphate synthase 1 N-acetyl glutamate H+ADP P₁ NH3 HCO3 ATP Carbamoyl Phosph. NH₂C-PO4 Ornithine 2. Citrulline formation P Citrulline C-NH₂ transcarbamoylase Aspartate NH₂ CYTOSOL MITOCHONDRIAL MATRIX https://canvas.uts.edu.au/assessment questions/356957/files/1562677/download? verifier=gRMPoy7VCgDrvn6QNfkZxDSsbLUwP1gRxFB3dLPj