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Which of the following preparations / drug group can not be expected to have positive effects on gastric ulcers?
Select an option:
Sucralfat
NSAID
Proton pump inhibitors
Histamine H2 antagonists
Prostaglandin analogues
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Solved in 3 steps
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- Why might a client be advised to avoid takingASA a few days before extensive oral surgery (e.g.,multiple tooth extractions)?Identify the side effects and special considerations associated with gastrointestinal system drug therapyIdentify the true statement about griseofulvin. Multiple Choice Griseofulvin is water rather than fat soluble. Less ultramicrosize drug may be taken on each dose compared to the microsized formulation. Ultramicrosize allows fewer particles to be in contact with the absorptive surface in the stomach. The clinical benefits derived from the conventional microsize formulation are less than the ultramicrosize formulation.
- Define the meaning of the term "peptic ulcer disease". Discuss some of the risk factors that make people susceptible (including factors that protect the mucosa of the stomach/duodenum and those that cause damage to it. ANSWER SHOULD INCLUDE: Accurate definition, e.g. mucosa break greater than 3-5mm in the stomach or duodenum with a visible depth. Discussion of factors e.g. infections (H. Pylori; others mainly in immunocompromised patients, e.g. cytomegalovirus ,tuberculosis, and syphilis), gastric bypass surgery, cigarette smoking, medication/drugs, physiological stress associated with critical illness (e.g. septicaemia), autoimmune diseases, eg, vasculitis, sarcoidosis, Crohn's disease. Discussion of factors e.g. protection: bucarb, blood flow, prostaglandin, mucus. Damage: H. Pylori, gastric acid, pepsin, drugs e.g. NSAIDExplain how an H2 antagonist agent affects gastricfunction.Match the terms in column A with the descriptions in column B. Column B1. activates protein-digesting enzyme trypsin2. causes emulsification of fats3. carries on phagocytosis in liver4. carbohydrate-digesting enzyme5. fat-digesting enzyme6. protein-digesting enzyme7. stimulates gallbladder to release bile8. stimulates pancreas to secrete fluids high in bicarbonate ions9. nucleic acid-digesting enzymeColumn Aa. amylaseb. bile saltsc. cholecystokinind. enterokinasee. Kupffer cellsf. lipaseg. nucleaseh. secretini. trypsin
- May you list the Oral mucosal absorption Formula relate to the surface area that drug being put on?Give a schematic diagram of how we can nonpharmacologically manage the Duodenal Ulcers ? Please answer at your own wordsDiscuss the physiologic influence of various pathologies, such as peptic ulcer disease, gastritis, spastic colon, gastroesophageal reflux disease, and hyperacidic states, on the health of patients and their gastrointestinal tracts