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When IkB is phosphorylated, NFKB is free to initiate gene expression promoting protein degradation. Studies suggest this pathway "exacerbates" in skeletal muscle. Aerobic exercise at a relatively low intensity may TNF-a. High intensity training may acutely increase TNF-a but may lower it long- term. Several studies have shown that both aerobic and resistance training may reduce inflammatic (TNF-a) and protein degradation (NFKB ) and reduce in response exercise. More specifically, acute bouts of exercise activate NFKB early in training while long-term training may down-regulate NFKB, promoting. Endurance training results in

When IkB is phosphorylated, NFKB is free to initiate gene expression promoting protein degradation. Studies suggest this pathway "exacerbates" in skeletal muscle. Aerobic exercise at a relatively low intensity may TNF-a. High intensity training may acutely increase TNF-a but may lower it long- term. Several studies have shown that both aerobic and resistance training may reduce inflammatic (TNF-a) and protein degradation (NFKB ) and reduce in response exercise. More specifically, acute bouts of exercise activate NFKB early in training while long-term training may down-regulate NFKB, promoting. Endurance training results in

Human Anatomy & Physiology (11th Edition)
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
Section: Chapter Questions
Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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When IKB is phosphorylated, NFKB is free to initiate gene expression promoting protein degradation. Studies suggest this pathway "exacerbates" in skeletal muscle. Aerobic exercise at a relatively low intensity may TNF-a. High intensity training may acutely increase TNF-a but may lower it long- term. Several studies have shown that both aerobic and resistance training may reduce inflammatior (TNF-a) and protein degradation (NFKB ) and reduce in response t exercise. More specifically, acute bouts of exercise activate NFKB early in training while long-term training may down-regulate NFKB, promoting Endurance training results in
Transcribed Image Text:When IKB is phosphorylated, NFKB is free to initiate gene expression promoting protein degradation. Studies suggest this pathway "exacerbates" in skeletal muscle. Aerobic exercise at a relatively low intensity may TNF-a. High intensity training may acutely increase TNF-a but may lower it long- term. Several studies have shown that both aerobic and resistance training may reduce inflammatior (TNF-a) and protein degradation (NFKB ) and reduce in response t exercise. More specifically, acute bouts of exercise activate NFKB early in training while long-term training may down-regulate NFKB, promoting Endurance training results in
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