The monoamine hypothesis of depression proposes that monoamine oxidase is overproduced in very depressed people. depression is a result of low monoamine levels at synapses. depression is a result of high monoamine levels at synapses. monoamine oxidase is absent in very depressed people.

Describe how the process of opioid withdrawal leads to a reduction in the release of dopamine?   Explain in a few sentences

Identify the multiple mechanisms by which a drug can be pro-ergic or anti-ergic at a synapse.

Briefly describe how silent glutamatergic synapses become activated during long term potentiation, describing all of the key neurotransmitters and their post-synaptic receptors and events that give rise to the process.

Would you consider caffeine a neurotransmitter? Provide reasons for your claim.

Which of the following changes would increase serotonin (5-HT) signaling between neurons? Select all that apply; partial point deduction for incorrect choices. O increase in serotonin synthesis O increase in the number of facilitating interneurons O decrease of serotonin receptors in the post-synaptic neuron inhibition of serotonin reuptake transporters O increase in activity of serotonin reuptake transporters O faulty transport and docking of synaptic vesicles decrease in serotonin synthesis O increase of serotonin receptors in the post-synaptic neuron