The higher the altitude, the lower the oxygen level in air. Climbers of very tall mountains risk altitude sickness, which is an illness characterized by shortness of breath, weakness, dizziness, and confusion. The early symptoms of cyanide poisoning are the same as those for altitude sickness. Cyanide binds tightly to cytochrome c oxidase, the protein that reduces oxygen molecules in the final step of mitochondrial electron transfer chains. Cytochrome c oxidase with bound cyanide can no longer transfer electrons. Explain why cyanide poisoning starts with the same symptoms as altitude sickness.

The higher the altitude, the lower the oxygen level in air. Climbers of very tall mountains risk altitude sickness, which is an illness characterized by shortness of breath, weakness, dizziness, and confusion. The early symptoms of cyanide poisoning are the same as those for altitude sickness. Cyanide binds tightly to cytochrome c oxidase, the protein that reduces oxygen molecules in the final step of mitochondrial electron transfer chains. Cytochrome c oxidase with bound cyanide can no longer transfer electrons. Explain why cyanide poisoning starts with the same symptoms as altitude sickness.

Human Physiology: From Cells to Systems (MindTap Course List)

9th Edition

ISBN:9781285866932

Author:Lauralee Sherwood

Publisher:Lauralee Sherwood

Chapter11: The Blood

Section: Chapter Questions

Problem 5TAHL

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The higher the altitude the lower the oxygen level in air. Climbers of very tall mountains risk altitude sickness which is characterized by shortness of breath, weakness, dizziness, and confusion. The early symptoms of cyanide poisoning are the same as those for altitude sickness. Cyanide binds tightly to cytochrome c oxidase, a protein complex that is the last component of the mitochondrial electron transfer chain. Cytochrome c oxidase with bound cyanide can no longer transfer electrons. Explain why cyanide poisoning starts with the same symptoms as altitude sickness.
Mutations that prevent Bax and Bak from interacting with the outer mitochondrial membrane would prevent the release of cytochrome c and inhibit intrinsic apoptosis. prevent the release of cytochrome cand promote assembly of the apoptosome. trigger the release of cytochrome c and promote intrinsic apoptosis. trigger the release of cytochrome c and promote the assembly of the apoptosome. trigger the release of cytochrome c and promote the cleavage of procaspase-8.
Which of the following components of the mitochondrial electron transport chain is the only one to be considered a peripheral protein? the cytochrome a-a3 complex the cytochrome c oxidase complex coenzyme Q (ubiquinone) the cytochrome b-c1 complex cytochrome c
Cyanide poisoning inhibits aerobic respiration at cytochrome c oxidase. Which of the following is NOT a result of cyanide poisoning at the cellular level? Select an answer and submit. For keyboard navigation, use the up/down arrow keys to select an answer. a b с d e Oxygen is reduced to water The rate of glycolysis increases Cells are forced to switch to anaerobic respiration The electron transport chain is not completed None of the above Answered K Open in Reading View ✔Posubmit
Which of the following is TRUE about cytochrome c protein? As a part of the ETC chain, it changes conformation to allow H+ ions to flow out of the matrix. It is found on the surface of the inner mitochondrial membrane and, therefore, contains start-transfer and/or stop-transfer sequences. With assistance from Bax, it can move out of the mitochondria and into the cytosol. It is considered a symporter because it is involved in electron transport. It is able to puncture holes in the plasma membrane to initiate apoptosis.
Which of the following components of the mitochondrial electron transport chain is the only one to be considered a peripheral protein? cell biology the cytochrome a-a3 complex the cytochrome c oxidase complex coenzyme Q (ubiquinone) the cytochrome b-c1 complex cytochrome c
b) Place a 'Yes' or a 'No' in the appropriate boxes that correspond to each listed feature of cellular respiration. You may need to state 'Yes' or 'No' in more than one box in a particular row in some cases. Cellular Respiration Feature Involved in aerobic respiration Occurs in the mitochondrial matrix Pyruvate molecules are produced Acetyl COA combines with a 4 carbon molecule Electrons are passed between protein carriers ATP is produced NAD+ gains hydrogen FADH₂ loses hydrogen Glycolysis Yes / No Krebs Cycle Yes / No Electron Transport chain Yes / No
Aerobic respiration in eukaryotes produces less ATP per glucose molecule than aerobic respiration in prokaryotes. This is because: the electrons from cytoplasmic NADH in eukaryotes are shuttled to ubiquinone instead of NADH dehydrogenase. prokaryotes can produce a larger proton gradient across the inner mitochondrial membrane, thus causing a greater driving force for ATP synthesis. fermentation and the tricarboxylic acid cycle can run simultaneously in prokaryotes, generating two additional ATP per glucose molecule. eukaryotes must actively transport glucose into the mitochondria where respiration А. B. C. D. occurs. prokaryotes can generate 2.5 ATP per FADH2 molecule, whereas eukaryotes can only generate 1.5 per FADH2 Е. E.
Fill in the blanks using the following words/acronyms: protons; Electron Transport Chain; oxygen; inner; Cytochrome c; NADH; ATP synthase; shuttle; ATP (note: the same word/acronym may be used more than one time) Complex I transfers electrons from to Coenzyme Q; and this transfer leads to the extrusion of from the mitochondrial matrix to the outside face of the mitochondrial membrane. Complex II allows the transfer of electrons from succinate to Coenzyme Q. Coenzyme Q is an electron which transfers electrons to Complex III. Electrons transfer from Complex III is associated with translocation. These electrons are then accepted by ; which transfers them to Complex IV. are also translocated at the level of Complex IV during electrons transfer; allowing the reduction of the final electron acceptor of the which is . ll the translocated are then channeled through the pore of the ; providing the driving force for release from the Enzyme complex.
Fill in the bold using the table. Because the inner mitochondrial membrane is impermeable to L, when there is an excess of mitochondrial L, it converts to M using a TCA condensation reaction, M crosses to the cytosol using a designated transport system. In the cytosol, M reconverts to L and oxaloacetate. Oxaloacetate is subsequently converted to malate with simultaneous production of N, a prerequisite cofactor for the glycolysis pathway. This reaction is then followed by conversion of N to pyruvate which produces O, a prerequisite cofactor for the fatty acid synthesis pathway. L M N O A. ATP pyruvate malate NADPH B. ATP citrate ATP NAD+ C. acetyl CoA pyruvate NAD+ NADH D. acetyl CoA citrate NAD+ NADPH E. glucose pyruvate malate NADP+
ak1: Why must the transfer of electrons from glucose to oxygen during aerobic respiration takes place in a series of steps? It is chemically impossible to transfer electrons directly from glucose to oxygen The energy of the electrons can be used to make ATP The energy of the protons can be used to make ATP The energy of the electrons can be used to make ADP
In order to pass the outer mitochondrial membrane a fatty acid Reacts with carnitine Is activated with coenzyme A Is activated with coenzyme Q Is oxidized