is to say, the excitatory pathways become over ive, or the inhibitory pathways, esigned to temper the excitatory pathways, are not active enough. Much of the research done on pilepsy focuses on voltage-gated sodium channels, and to date over 700 different mutations to the hannel have been identified as playing a role in epilepsy. The means by which these mutations ontribute to epilepsy is quite complex. Let's simplify and apply what we have learned so far to dentify potential mechanisms for this condition. what way could voltage-gated sodium channels be affected in excitatory neurons which would ncrease the likelihood of the neuron firing an action potential? (one correct answer) The inactivation gate is slower to close. The channel takes longer to reset to its original position ('ready state'). The channel is activated at a more negative membrane potential. The channel has a lower permeability to Na* compared to normal. Any of the above mechanisms could make the cell more likely to fire an action potential.

Define monera.

Give typing answer with explanation and conclusion

Give an example of a specific function of necessary for life or reproduction in the human that is maintained by a feedback loop. Describe the system, giving the stimulus, the response, and how that maintains function of the system. describe a real or hypothetical condition caused by disruption of this system.