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- People with galactosemia display central nervous system abnormalities even if galactose is eliminated from the diet. The precise reason for it is not known. Suggest a plausible explanation.Glucokinase acts as a glucose sensor in hepatocytes (livercells), a- and b-cells in the pancreas, enterocytes (intestinalwall cells), and the hypothalamus (a control center in thebrain of numerous physiological processes). Explain whyglucokinase can perform this role.One metabolic scenario that leads to insulin resistance is the elevation of the conccentration of free fatty acids in skeletal muscle cells. Would you expect increased activity of diacylglycerol acyltransferas (DGAT) to reduce or exacerbate the resistance? Explain.
- Although animals cannot synthesize glucose from acetyl-CoA, if a rat is fed 14C-labeled acetate, some of the label appears in glycogen extracted from its muscles. Explain.Animals cannot carry out a net conversion of even chain fatty acid carbon to glucose. However some of the carbons in odd-chain fatty acids can be gluconeogenic precursors to glucose. Explain.Glucagon The function of glucagon is in promoting energy release and inhibiting energy storage in response to low circulating glucose levels, which are largely controlled by insulin [24]. Besides its role in blood–glucose regulation, patients treated with glucagon receptor an tagonists (GRA) often suffer from dyslipidemia, fatty liver and weight gain, suggesting that glucagon play an important role in lipid metabolism [25]. Glucagon acts mainly on the hepatocytes which possess the highest level of glucagon receptors. Following mecha nisms may be involved during this process: (1) At the molecular level, following glucagon binding to its receptor on the hepatocyte, cAMP will be activated and accumulated, which in turn activates cAMP-response element-binding protein (CREB). As a result, the tran scription of carnitine acyl transferase (CPT-1) increases, which converts fatty acids into acylcarnitine and activates β-oxidation-increasing fatty acid catabolism [26]. In addition,…
- The interconversion of DHAP and GAP greatly favors the formation of DHAP at equilibrium. Yet the conversion of DHAP by triose phosphate isomerase proceeds readily. WhyWhen the identical subunits of chicken liver fatty acid synthase are dissociated in vitro, all of the activities can be detected in the separated subunits except for the β-ketoacyl synthase reaction and the overall synthesis of palmitate. Explain these observations.What is an example of a biomolecule that interacts with Insulin? Please give a detailed explanation of that molecule's structure as well!
- How many ATP are produced from the complete degradation of 1,3-bisphosphoglycerate? the complete degradation of ONE glucose using the malate-aspartate shuttle as your guide.Draw the products that would be produced from phospholipase C cleavage of a glycerophospholipid with a phosphoserine head group, an alpha linoleic acid attached to C2 of glycerol and a linoleic acid attached to C, of glycerol. Draw these products at pH=1. Be sure to number the fatty acid carbons in your products.Lithium ion inhibits the synthesis of inositol trisphosphate by inhibiting a reaction in the breakdown of inositol trisphosphate.Explain this apparent paradox.