(ii) Calculate the minimum length of the DNA base sequence required to code for titin Show your working.

Inside of skeletal muscle cells are voltage-controlled calcium ion (Ca2+) gate proteins. These are integral for the contraction mechanisms of skeletal muscle. A picture of them is shown below.   These are regulated by voltage, as indicated above. How would voltage serve to open or close these gates?     Voltage is a component of hydrogen bonds, which is necessary to maintain the secondary structure of the protein; without voltage, the protein starts to chemically degrade.   Voltage actually does not serve as the direct mechanism of opening or closing gated proteins.   Changing the voltage changes whether the R groups are charged or not, which alters the actual primary structure of the protein   Changing voltage changes the nature of the tertiary structure, causing the protein to change its shape to open or close.

You are a brilliant (but evil) biochemist who is developing a toxin that can be used to paralyze skeletal muscle. Using your knowledge of the sequence of cellular/molecular events that cause a muscle contraction, identify two parts of the process that could be disrupted to cause paralysis, and explain the specific effect of each disruption on contraction. please help

Hi, I only need the answer for this particular question, if that's ok: The swo-1 a mutation leads to worms with more body wall muscle, whereas worms with the swo-1 b mutation are not able to move. Based on these phenotypes and the findings from questions 3 and 4, describe the role thewild-type version of this protein plays in muscle function. Thank you!