How would the ATP yield per glucose molecule be different if enzyme specificity forced FADH2 to be used in place of NADH throughout aerobic metabolism?

Processes taking place in the mitochondrion convert the chemical potential energy of NADH into the chemical potential energy of ATP. A proton concentration gradient (Δ[H+]) is part of this process. Describe exactly where this gradient is located, and how the gradient is produced. In terms of the gradient, where is the higher [H+] and where is the lower [H+]?

The following Michaelis-Menten plot shows the response of phosphofructokinase-1 (PFK-1), an enzyme in the glycolytic pathway, to the presence of ATP and AMP. Based on the data below, which of the following statements apply? Select all that apply. No inhibitors (low (ATP)) 1 mM ATP + 0.1 mM AMP 1 mM ATP 1.0 2.0 (Fructose-6-phosphate) mM Fig. : Regulation of PFK activity AMP is an allosteric modifier that enhances the binding of the substrate fructose 6-phosphate а. O b. AMP is an allosteric modifier that reduces the binding of the substrate fructose 6-phosphate ATP is an allosteric modifier that reduces the binding of the substrate fructose 6-phosphate O c. O d. ATP is an allosteric modifier that enhances the binding of the substrate fructose 6-phosphate Phosphofructokinase activity

Identify the steps in the glycolytic and citric acid cycle pathways where ATP, GTP, NADH, and FADH2  are produced.