Explain why the degradation of odd-chain fatty acids can boost the activity of the citric acid cycle.

Why is it that the carboxylate "ions" actually partake in the citric acid cycle ratherthan the carboxylic acid? Explain using an example

This is a depiction of the connection between the urea cycle, the malate-aspartate shuttle, and the citric acid cycle. In this depiction, blank boxes with numbers are hiding the name of a metabolite. Using the list below the pathway name the missing metabolites. Hint: Start by identifying #1, #6, and #7 and work from there.

Describe the mechanism for moving acetyl-CoA produced in the mitochondrial matrix into the cytosol for fatty acid synthesis.

how many net ATP molecules does the citric acid cycle generate in total? Consider both ATP generated directly by the citric acid cycle and ATP generated downstream in oxidative phosphorylation using other molecuies generated by the citric acid cycle For your answer, consider ATP and GTP to be equivalent A) 32 B) 20 C) 2 D) 10 E) 1

Describe how the citric acid cycle is regulated

Discuss the relationship between the pentose phosphate pathway and glycolysis.

Why is it reasonable for excess NADH to negatively regulate the citric acid cycle?

a) What carbon-containing molecule exits the citric acid cycle?  Why doesn’t your body capture the carbon-containing molecule you identified in part (a) and use that molecule to build new sugars and fats? Explain your answer.  You have just created a new enzyme that will make the citric acid cycle more efficient! Instead of the citric acid cycle taking 8 steps, all of the bonds between the carbon atoms will be broken in only one step! Is this a good idea? Explain your answer.

The two main goals of the citric acid cycle are:   (a) citrate synthesis and gluconeogenesis (b).degradation of acetyl-CoA to produce energy and supply precursors for anabolism (c).degradation of pyruvate to produce energy and supply precursors for anabolism (d).degradation of glucose to produce energy and supply precursors for anabolism (e).degradation of pyruvate to produce energy and synthesize oxaloacetate for gluconeogenesis