Concerning neurons, DESCRIBE how local anaesthetics prevent the propagation of an Action Potential.

starting with an axon that is maintaining a resting potential, describe the sequence of events that causes the generation of an action potential, including the electrical charges and ions involved.

Describe how an action potential is generated in a neuron.  Include channels that are open/closed during this process and be sure to include terms such as depolarization, threshold, hyperpolarization, and repolarization.

In SA nodal cells the upstroke of the action potential:A)is characterized by a substantial overshoot component.B)results from rapid inward movement of Na+.C)results from inward movement of Ca2+D)results from inward movement of K+.

Give examples of graded potential, resting potential and action potential  seen in neurons.

Discuss the importance of the sodium-potassium pump in the establishment of resting potentials and the initiation of action potentials in neurons.

Explain the role of ion channels and sodium-potassium pumps in the establishment of the resting potential and the generation of an action potential.

With respect to the three structural types of neurons (unipolar, bipolar & multipolar): State which parts of each type of neuron receive information, which parts integrate information, and which parts conduct the output signal of the neuron. Describe the location of the cell bodies of each type of neuron within the nervous system.

Explain the importance of neurotransmitter removal from the synaptic cleft and describe three removal mechanisms.

What type of synaptic potential (if any; be sure to indicate if any modification is occurring as well) would occur if: a.) An MAO inhibitor type of antidepressant is functioning at an active dopaminergic synapse that increases the rate of sodium flowing into the cell. b.) Prozac is present at an active serotonin synapse where receptor activation increases the flow of potassium out of the cell.