Binding of neurotransmitter to the motor end plate leads to depolarization because the neurotransmitter: a) opens ligand-gated sodium channels. b) opens voltage-gated sodium channels.

The axon of neurons contains microtubules with the minus end situated toward the cell body and the plus end situated toward the end of the axon.  Neurotransmitters are transported in vesicles carried by motor proteins.  A neuron missing which of the following motor proteins would be unable to secrete neurotransmitters in this way? Group of answer choices: Myosin Kinesin Dynein

Place the events at the neuromuscular junction in the order in which they occur: 1. release of acetylcholine (ACh) 2. activation of ACH receptors on the motor endplate 3. termination of the ACh activity by acetylcholinesterase 4. arrival of an action potential at the synaptic end bulb 5. diffusion of ACh across the synaptic cleft 6. opening of NA* channels in the motor endplate 7. production of a muscle fiber action potential O 4,1,2,5,6,7,3 4,1,5,6,2,7,3 4,1,5,2,7,6,3 4,1,5,2,6,7,3

Caffeine is known to alter transmembrane movement of calcium. What are two possible sites in the nerve and/or muscle that caffeine can act upon?

1. If you block calcium channels on an axon, which will not occur? A) exocytosis of neurotransmitter B) repolarization phase of the action potential C) depolarization phase of the action potential D) hyperpolarization phase of the action potential E) graded potential

During exercise, activation of alpha-adrenergic receptors in skeletal musclearterioles:A)is opposed by metabolic vasodilation.B)decreases rapidlyC)causes relaxation of the vascular smooth muscle.D)causes hyperpolarization of vascular smooth muscle.E)causes activation of stretch receptors on the endothelium

Which of the following causes ( the state of ) rigor in skeletal muscle ?a) lack of atp b) decrease of intracellular ca2plus c) increase in intracellular ca2+ d) lack of action potential in motor neurons