An individual has a genetic deficiency that prevents the production of glucokinase. Following a carbohydrate meal, do you expect blood glucose levels to be high, low, or about normal? What organ accumulates glycogen under these circumstances?

How does a ketogenic diet decrease the level of urea in obese patients and increase the level of creatine in obese patients? (explain on a molecular level)

Excessive breakdown of fatty acids produces large amount of acetyl CoA that result in the production of ketone bodies. Is that a healthy condition or harmful to the body?

How are glucose and glycogen release regulated in the body?

Barry is participating in a nutrition study in which he must follow a prescribed diet and have his blood tested regularly. On one occasion, his test results indicate low levels of insulin, elevated levels of glucagon, and epinephrine and moderate levels of cortisol and growth hormone. What would you expect his levels of glucose, ketones, and fatty acids to be? Explain the rationale behind your answers.

Name the hormone responsible for converting glycogen to glucose.

Name the protein that is up-regulated after a high energy content meal in an effort to increase energy expenditure to prevent the development of obesity.

What are the end-products of the complete aerobic catabolism of glucose?

Glucose 6-phosphatase deficiency is a glycogen storage disease referred to as von Gierke disease. Which of the following symptoms would you NOT expect to see in patients with this disease? (A) severe fasting hypoglycemia (B) excess adipose triglyceride storage and low circulating levels of fatty acids (C) hyperammonemia (D) liver enlargement resulting from excess liver glycogen storage (E) lactic acidosis (F) gout

Gluconeogenesis is the process for the synthesis of new glucose from the non-carbohydrate precursors. Select one: True False