The Causes And Complications Of Devil Facial Tumour Diseases

This form is the least dangerious out of the skin cancer forms. This one grows very slow but it is located on your head, neck and upper torso.this can appear to be on dry or lumpy on yourskin and can appear to be scaly. This is red,pearly and pale looking in color. This can form to be a sore that wont heal properly on you skin.

were covered in mysterious black boils that oozed blood and pus. That is why they gave the

Tasmanian devils have been at the top of the Australian predators. They are one of the deadliest animals in Australia. They are the size full a full grown furry cat. They are full black and have a white stripe in front of their chest. They mainly feast on meat, they are carnivores. They eat snakes and little animals. From eating the tiny rodents and animals, one major downfall has come into play, the Devil Facial Tumor Disease.

This syndrome is from a mutation of a gene on chromosome 15 and this causes problems in the production of fibrillin-1 which is a protein that is an important part of connective tissue. The name for the gene is FBN1. Basically, it is the “glue” that helps to support the tissues in the human body. A child born to a parent with this syndrome has a 50% of having it. However, in the remaining 25%, neither parent has the disease which gives them a 1 in 10,000 chance of having a child with this disorder. When a child of two unaffected parents is born with it then the genetic mutation occurs in either the egg or sperm cell at the time of conception.

The breakdown of the myelin sheath is caused from a mutation of the gene that makes the Adrenoleukodystrophy protein (ALDP). This ALD protein helps the body metabolize saturated very-long-chain fatty acids found in the serum and tissues of the central nervous system. The newly mutated gene no longer acts as a help aid to breaking down the long-chain fats. Therefore, the body starts accumulating an abnormal amount of fat in the nervous system, adrenal gland and testes that sets off an unusual response in the immune system; demyelination.

The ability to recognize SSWD in its early stages is detrimental to differentiating healthy and infected stars, however the early stages can be difficult to detect even for a trained eye. To better analyze the rate of SSWD researchers distinguish the symptoms by categories of infection. Category 1 consists of small lesions on the limbs that can be mistaken for scars or wounds from a bird, this is the stage that is most often overlooked. Following small wounds the internal organs will emerge from the lesions followed by the loss of one or more limbs. The final category results in the loss of three or more limbs and the eventual death of the organism. Researchers document the species affected as well as the severity and local population density to determine the species most impacted by the

Dzevdet Smajlovic, professor from the Department of Neurology at the University of Tuzla in Bosnia stated that

Tasmanian Devils are predators and are near the top of the food chain. They are about the size of a cat with furry hair. They eat birds, snakes, fish and insects and survive, actually quite well. But a disease is spreading across the species. It is called the "devil facial tumor disease". Also referred to as DFTD.

Tasmanian devil (Sarcophilus harrisii) populations on the island of Tasmania have experienced a rapid decline during the past twenty years due to the spread of a cancer called Devil Facial Tumor Disease or DFTD. DFTD is a deadly contagious cancer that is characterized by red oozing lesions or tumors that form on the face and mouth of the Tasmanian devil. The cancer spreads from one devil to another when a DFTD infected devil bites a healthy devil thereby infecting the open wounds with cancer cells. Once contracted the devil dies within six months due to infection or starvation because the tumors in mouth hinder feeding. However, researches lead by biologist Andrew Storfer have discovered that some Tasmanian devil populations have evolved a

Neurofibromatosis is a genetic disorder that causes tumors to form in the brain, spinal cord, and nerves. There are 3 types of this disorder. Type 1 usually appears in childhood, while type 2 and 3 appear in early adulthood. Type 3 can cause chronic pain throughout the body. Some cases may not require treatment other than careful observation. Other cases may require chemotherapy, radiation therapy, or surgery. In this essay i will explain the causes of the disorder, symptoms, treatment, and a day in the life of that person

Cancer is a disease that over the years seems to occur more and more amongst, not only the human race, but animals as well. Signs of cancer are not always evident and can go unseen.

Frontotemporal Dementia, also known as Frontotemporal Lobar Degenerations, was first found and described by Arnold Pick, M.D., in 1892. This disorder is a broad term used to characterize multiple uncommon disorders that disturb the frontal and temporal lobes of the brain. These areas of the brain are linked to personality, language, and behavior; Pick was able to diagnose the first FTD patient due to the noticeable symptoms that affected speech and language (Alzheimer’s Association, Alz.org). Fragments of the frontal and temporal lobes deteriorate and this is what causes Frontotemporal Dementia to become a serious problem.

Multiple Sclerosis is an autoimmune disorder where the myelin sheath within the Central Nervous System is attacked (National Multiple Sclerosis Society, 2017). The myelin sheath protects the axon of the nerve cell. When the myelin sheath is intact, the axon is able to carry impulses away from the neuron’s cell body, and the message carried is clear. With Multiple Sclerosis, the myelin sheath becomes scarred, hence the word “sclerosis”, and distorts the nerve impulses traveling over the CNS (National Multiple Sclerosis Society, 2017). This may cause the message to be changed or stopped altogether.

Affecting women more than men, Multiple Sclerosis (MS) is normally diagnosed between the ages 20 and 40, but can be seen during any age.; which is the most common disabling neurological disease of young adults. It’s caused by the myelin sheath, which is the protective covering which surrounds the nerve cells, by being damaged. Nerve signals start slowing down or they stop when this nerve covering is damaged, which is caused by inflammation. It can affect any area of the brain, spinal cord, and optic nerve. The causes for Multiple Sclerosis are still unknown, however the common thought on it is that it’s a virus or gene defect, and environmental facts could also play a role in it. With MS, it causes the sheath to be stripped away, which is a process called demyelination. Severe or mild disabilities can occur depending on the nerves that were affected. If the myelin in the sensory nerves is lost, a person may have impaired sense of touch.

Multiple sclerosis one a nervous system disease that affects the brain, spinal cord, and the optic nerves in the eyes. It causes problems with vision, balance, muscle control, and other basic body functions. The effects are often varying from person to person (“Multiple Sclerosis”, n.d). MS happens when the immune system attacks a fatty material named myelin, that protects the nerves. Without the protection of this outer shell, the nerves become damage. Scientists and doctors studied for centuries what multiple sclerosis is and they were unable to define it for long period time “MS has been conceived from the 14th century through the early 20th century (Butler, 2003). During those periods, the physicians and scientists faced hardship to distinguish between MS and other cognitive diseases. During the 21 century, because of technological advancement and researches, MS studied well and consequently treatment options reflected in a new belief system (Butler, 2003).