S.D. is a 45-year-old woman who comes to the ED complaining of sudden onset of palpitations and shortness of breath. Standard protocol requires you to obtain a 12-lead ECG and attach S.D. to the cardiac monitor for continuous monitoring. A 12-lead ECG recording for S.D is ordered while you print out a rhythm strip from the cardiac monitor.
1. Describe the appropriate location to apply the leads for both the 5-lead cardiac monitor as well as the 12- lead ECG.
- For the 5-lead cardiac monitor, there are five electrode pads placed. The RA electrode (white) is placed below the right clavicle (2nd interspace, right midclavicular line), the LA (black) is placed below the left clavicle (2nd interspace, left midclavicular line), the RL ( green) is
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5. What treatment might you expect the health care provider to initially order for S.D.’s atrial fibrillation?
- The goals of treatment include a decrease in ventricular response (to less than 100 beats/minute), prevention of stroke, and conversion to normal sinus rhythm, if possible. To accomplish this, I would expect the health care provider to initially order drugs to control the ventricular rate, such as calcium channel blockers, beta-adrenergic blockers, digoxin, and dronedarone. For some patients, pharmacologic or electrical conversion of the atrial fibrillation to normal sinus rhythm may then also be considered, such as by using amiodarone or electrical cardioversion. If the atrial fibrillation lasts for longer than 48 hours, anticoagulation therapy will be needed for 3-4 weeks before the cardioversion and for weeks after as well. If drugs or cardioversion do not work, radiofrequency catheter ablation and the Maze procedure would be expected as further options.
S.D. was admitted to the telemetry unit and an IV amiodarone drip was started. The purpose of the drug was to convert her atrial fibrillation to normal sinus rhythm. Although her heart rate has decreased to 108 beats/minute, she remains in atrial fibrillation 24 hours later. A cardiologist was consulted and electrical cardioversion is
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What specific ECG change will you be looking for to determine if S.D.’s chest pain is related to cardiac ischemia? Injury? Infarction?
- To determine if the patient’s chest pain is related to cardiac ischemia, you would look for ST-segment depression and/or T wave inversion. If the ST-segment depression is at least 1mm (one small box) below the isoelectric line, it is significant and occurs in response to inadequate supply of blood and oxygen, which leads to an electrical disturbance. Once this is treated, adequate blood flow is restored, the ECG changes will resolve, and the ECG will return back to patient’s baseline.
To determine if the patient’s chest pain is related to injury, you would look for ST-segment elevation. Myocardial injury represents a worsening stage of ischemia. If ST-segment elevation is greater than or equal to 1mm above the isoelectric line, it is significant and treatment needs to be prompt and effective to try to restore oxygen to the myocardium, and to avoid or limit infarction. The absence of serum cardiac markers confirms that infarction has not
12 Lead Electrocardiogram (ECG) - There are typical changes to the normal pattern of the ECG in a heart attack. Patterns that occur include pathological Q waves and ST elevation (Koutoukidis, Stainton & Hughson 2013, p. 505). However, it is possible to have a normal ECG even if a patient has had a heart attack. The indicators for this test include: suspected myocardial infarction, suspected pulmonary embolism, perceived cardiac dysrhythmias, fainting or collapse, a third heart sound, fourth heart sound, a cardiac murmur or other findings to indicate structural heart disease. The
Mr. S was driving when he experience a stabbing chest and back pain for the first time. The pain was so severe he immediately went to his local ER. Pulmonary ventilation and perfusion (VQ) scan and Computed tomography angiography (CTA) was done at his local ER. VQ scan was negative for pulmonary embolism (PE). CTA of the chest revealed
On arrival at the ED, the physician auscultates muffled heart tones, no breath sounds on the right, and faint sounds on the left. A.W. is
In the evaluation of patients with chest pain, the preliminary ECG is a more clear-cut tool for early risk stratification with more recent recommendations indicating that ECG should be performed as early as possible, within 10 minutes of ED admittance. Early indicators associated with MI or ischemic complication such as ST segment elevation or depression allows rapid treatment aligning with the indicated complication. While the ECG may reveal significant indicators in certain situations, in other circumstances findings may be limited due to low diagnostic sensitivity
Parker included collecting cues in 12-lead ECG, blood pressure, potassium level, sodium level, the warmth of hands and feet and pain score (Corrales-Medina et al., 2012). Also, the process involved identifying the risk factors associated with the patient's condition. From the immediate assessment, it included weight, smoking history, history of depression and family history of cardiac conditions (Corrales-Medina et al., 2012). One evening, Mr. Parker slumped on the bed; I monitored the continuous cardiac monitor to determine his heart rhythm. Based on current information, the T wave on the ECG indicated that the ventricles are repolarising (Levett-Jones et al., 2010).
ECG : ventricular rate 54 beats/min, HR varying from 39 to 60 during a 45 minute period of monitoring, infrequent PVCs, ST elevation in leads II, III and avF indicating inferior injury or ischemia secondary to acute MI.
Each of these monitors is designed to alarm nursing staff whenever a drastic change has occurred with a patient’s condition. For example, if a patient’s heart rate drops below sixty beats per minute then an alarm will sound to alert the nurse that the patient has become bradycardic. These alarms are intended to protect the patient from harm and ensure that they receive timely interventions from nursing staff and physicians (AAMI, 2011).
Cardiac dysrhythmias come in different degrees of severity. There are heart conditions that you are able to live with and manage on a daily basis and those that require immediate attention. Atrial Fibrillation is one of the more frequently seen types of dysrhythmias (NIH, 2011). The best way to diagnosis a heart condition is by reading a cardiac strip (Ignatavicius &Workman, 2013). Cardiac strips play an chief part in the nursing world allowing the nurse and other trained medical professionals to interpret what the heart is doing. In a normal strip, one can clearly identify a P wave before every QRS complex, which is then followed by a T wave; in Atrial Fibrillation, the Sinoatrial node fires irregularly causing there to be no clear P
This report is an analysis of an anonymous patient’s medications and how they relate to his health issues. Please note that the patient will be referred to as patient Afib in order to protect his privacy. Patient Afib is a 58 year old male with a recent onset of atrial fibrillation (AF) that has been cured using ablation and antiarrhythmic medications. This report discusses how physicians treated patient Afib’s AF, the medications patient Afib is currently taking and their mechanism of action, and how these medications relate to the patient’s disease. Medications discussed in this analysis include Proair HFA, Multaq, Toprol XL, and Coumadin. Proair HFA, a rescue inhaler used to treat asthma, was prescribed to patient Afib in order to determine if AF or underlying asthma caused his shortness of breath. Multaq is an antiarrhythmic drug used to keep patient Afib in a normal sinus rhythm. Toprol XL is a medication used in the treatment of hypertension that also exhibits rate controlling properties which prove beneficial for patient Afib. Coumadin, an anticoagulant, is used to reduce the risk of stroke for patient Afib should his AF ever spontaneously return without his knowledge. Patient Afib does not like the regime of pills he is required to take but understands their necessity.
There are three types of atrial fibrillation. The first type is paroxysmal atrial fibrillation. It will occur at random time and stop by its own. This type of AF would normally stop within one week and it does not require any treatment. It will normally cause the most symptoms in people who has it as it is so unpredictable and
A recent study shows 1:4 American adults over the age of 40 are at risk of developing an abnormal heart rhythm (www.myvmc.com). Once an arrhythmia has been diagnosed an antiarrhythmic agent can be prescribed. It is important that when taking an antiarrhythmic medication, directions or how it should be taken is followed precisely. As long as the antiarrhythmic agent is taken as prescribed, they will help prevent further complication, such as heart attacks and heart
The low mortality rate observed in patients without heart disease, even in the absence of an effective therapy, makes it difficult to demonstrate any variation in the prognosis. The basic objective in these patients is to improve symptoms and quality of life, to avoid the adverse effects of antiarrhythmic agents and to cure the arrhythmia in order to prevent the development of atrial dilation, need for anticoagulant therapy and increased risk. In population-based studies, as the Framingham study, the presence of AF increased long-term mortality 1.5 to 1.9 times.
Once at the hospital tests will be done to rule out other chest pain related causes. The first test that will be done is an Electrocardiogram or an EKG, which records the hearts electrical activity. Damaged heart cells are not able to produce electrical impulses which will produce abnormal EKG results. Elevations in the ST waves on an EKG are classified STEMI and are present in over ninety percent of myocardial infarctions who had a complete occlusion to an artery (Cardiac Emergencies, n.d.). NSTEMI is where there is no elevation of the ST wave and is indicative that a full occlusion has not occurred (Cardiac Emergencies, n.d.).
The ECG is a test that connects wires to the chest and arms displaying the electrical signals of the heart on a monitor. In atrial fibrillation, the monitor will display no discernable, independent P waves, but rather replaced by evident F waves. The QRS complex will vary with R-R intervals and result in a rapid, narrow complex (Goralnick, 2015). The ECG can also provide other information such as presence of bundle-branch block, left ventricle hypertrophy, and prior myocardial infraction (Floyd, 2016). The holter monitor is a portable ECG that is carried around and records 24 hours or more of heart activity to later be interrupted by the doctor. The event recorder is again the portable ECG that is intended to record weeks to months of heart activity and records only if an episode of atrial fibrillation occurs. The echocardiogram is a noninvasive test that shows a video image of the heart originated by sound waves. These images can show if there is any structural damage of the heart. Blood tests are completed to eliminate thyroid issues or other biomarkers in the blood that could be causing the atrial fibrillation (Mayo Clinical Staff, 2015). Positive biomarker results are elevated C-reactive protein and B-type natriuretic peptide
Finally, ensure that the patient is supine on a bed, comfortable and relaxed. Cowley (2002) describes how accurate chest lead placement is essential for ensuring quality ECG output, as any misplaced leads may result in a change in ECG waveform, in turn this may cause the ECG trace to be misinterpreted. The full pictorial description