Gastritis Research Paper

According to Fitzgerald, Helicobacter Pylori bacteria is found in 90% of the patient with duodenal ulcer (2015). This organism produces beta lactamase, therefore it’s resistance. Patient will need an antimicrobial therapy to eliminate the H. pylori infection and prevent recurrence of the disease in addition to an acid suppression therapy. The treatment therapy is known “standard triple therapy” That includes Proton pump inhibitor as well as clarithromycin and amoxicillin or metronidazole (Das, Sureshkumar, Sreenath & Kate, 2016). If the disease is caused from repeated use of NSAID then the drug of choice is Misoprostol 200mcg four times daily with food until NSAID is discontinued. Another form of treatment if cause does not relate to NSAID

Helicobacter pylori, also known as H. pylori, is a spiral bacterium that lives inside the human stomach. H. pylori likes the acidic environment that the stomach provides and evidence has linked it to a variety of minor gastrointestinal illnesses. H. pylori causes inflammation in the lining of the stomach and is involved in 90% of stomach and intestinal ulcers. H. pylori also has an apparent cause to the development of adenocarcinoma, a common stomach cancer. J. Robin Warren first detected H. pylori in the stomachs of ulcer patients in 1979. Warren then isolated the microbe in a culture and tested it effects by swallowing some of the inoculum. A short-term case of gastritis (inflammation of the lining of the

Antacids, like Tums, may treat the symptoms. Proton pump inhibitors are a group of drugs that stop the production of gastric acid. Histamine 2 receptor antagonists are used to block the action of histamine on parietal cells in the stomach by decreasing the production of acid by these cells. These two medications help to treat GERD. (Davis, 2011). Any age group can take these medications, and the dose is prescribed by the doctor. Acceleration of gastric emptying is managed by prokinetics. If medication cannot resolve the GERD, then surgery may be necessary. One type of surgery is a Fundoplication. This is when a wrap to the stomach fundus is made around the distal esophagus to restore the competence of the lower esophageal sphincter. If it is needed, a gastrostomy can be performed at the same time as the Fundoplication if decompression of the stomach is needed after the surgery. For the child who has problems with gastric emptying, another procedure is a pyloroplasty with the Fundoplication. Silvestri, 2011). The nurse will have to monitor the patient for abdominal complications after surgery like bleeding, and pain management. The child will have to start out with clear liquids and slowly move toward a regular diet. Instruct the parents to avoid giving their child a heavy solid

Explanations for the fast rise in occurrence of EAC involve the increasing prevalence of gastroesophageal reflux and obesity as well as the declining incidence of Helicobacter pylori infection. Helicobacter pylori infection is correlated with hypergastrinemia, the excess of gastrin in the blood. Gastrin is a hormone that is secreted by G cells situated in the gastric antrum and works on the parietal cells to trigger the production of hydrochloric acid. Constant hypergastrinemia due to daily PPI therapy contributes to the raised gastric acid-secretory capacity that is not obvious throughout PPI therapy, but that emerges quickly when the drug is stopped. Increased acid production due to hypergastrinemia poses a risk factor for development of

A gastric injury model based upon a modification of the method described by Mizui and Doteuchi 15 was induced by acidified ethanol solution (150 mMHCl/absolute ethanol) 40:60v/v, (HCl/ethanol solution). Normal control groups were orally administered Normal saline. Ulcer control groups were orally administered normal saline . Reference group received oral doses of 20 mg/kg omeprazole. Experimental groups were orally administered with Sanguparpam 100,200,300 mg/kg. One hour after this pretreatment, normal control group was orally administered with normal saline, and HCl/ethanol solution (5 mL/kg) was orally administered to ulcer control group, reference group, and experimental group in order to induce gastric ulcers. The rats were euthanized 60 minutes later 16 under an overdose of xylazine and ketamine anesthesia and their stomachs were immediately excised. Ulcer index was determined. The excised stomach was used to evaluate gastric wall mucus, antioxidant enzymes ,lipidperoxidation, Myeloperoxidation, prostaglandin estimation and Histopathological

• Some physicians administer gastric and duodenal ulcer prophylaxis in the form of a proton pump inhibitor (PPI) if the patient has a history of gastrointestinal ulceration

Ulcerative colitis is a chronic condition which is part of inflammatory bowel disease. It causes inflammation (swollen) in the GI tract. Usually swollen occurs in the large intestine (colon) and rectum. In this disease small sores (ulcer) develop on the innermost part of the colon and those ulcers produce pus and mucus. Because of the pus and mucus patient may feel abdominal discomfort.

b).Use of nonsteroidal anti-inflammatory drugs such as aspirinor ibuprofen, which can cause ulcers and gastritis

One common cause of gastritis is H. Pylori. H. Pylori is a bacteria found worldwide, but especially in developing countries, where up to 10% of children and 80% of adults are likely to have had an H. pylori infection. However, patient usually are without any symptoms (Pilotto & Franceschi, 2014). The human host is the only known reservoir. Transmission occurs by person-to-person contact, by both fecal-oral and oral-oral routes therefore, infection is most likely acquired in childhood (Go,2003).

Stress-related mucosal disease (SRMD), an acute erosive gastritis that may potentially lead to stress ulcers and/or stress-related injuries, remain a significant concern for patients within the critical care setting.1 The initiation of anti-secretory therapy (AST) for stress ulcer prophylaxis (SUP) within the critical care setting has gained widespread use as it has been noted to protect against SRMD and clinically important gastrointestinal (GI) bleeding.2 Although the use of these agents is well established in the critical care setting, several research studies have confirmed the extensive and inappropriate use of AST in both intensive care unit (ICU) and non-ICU patients, based on current recommendations.3

The nonpharmalogical treatment of peptic ulcer disease involves eliminating the use of NSAIDs, ceasing smoking, and avoiding foods and beverages that could irritate the ulcer such as spicy foods, caffeine, and alcohol.1 The patient should take non-NSAIDs for the treatment of pain such as acetaminophen. The recommended treatment regimen for patients with NSAID-induced gastric ulcers involves either a proton pump inhibitor, an H2 receptor

This study employs a retrospective study design to identify any association between upper gastrointestinal disease among college student and their demographics as well as lifestyle, including status at UCF, major, smoking, alcohol intake, diet, nonsteroidal anti-inflammatory drugs (NSAIDs), stress, and sleep. Participants were recruited from the University of Central Florida, a survey was built though SurveyMonkey and distributed to students at the University of Central Florida via email. The survey was utilized as the primary data collection method. The survey included questions on demographics, general health, smoking, alcohol intake, stress, and sleeping habits. It also included an Izumo scale questionnaire to measure the symptoms of gastrointestinal problems, the Izumo Scale was previously tested for validity. This sample included all enrolled students at UCF at the time of survey distribution during Summer 2016, consequently the overall participation of 1475 was large enough to be representative of UCF. The study was submitted to, an approved by the University Institutional Review Board (IRB), the survey (Appendix-1) was distributed via mass email, and was open for four weeks. The purpose of this study is to report any association between gastrointestinal symptoms among college students, using the Izumo Scale. All collected data from SurveyMonkey was downloaded into SPSS and corresponding data was descriptively analyzed and some was analytically

As a child, I had the habit of drinking coffee constantly in addition to eating spicy food daily. One day I woke up with an ulcer, and having the tendency to vomit. My parents took me to the clinic after 3 days of having the stomach pain because my sickness did not heal on its own. The doctor diagnosed that I had gastritis, which is the inflammation of the lining of the stomach. In the moment of the diagnosis, he did not explain what gastritis was, but explained that the coffee intake was the cause of my gastritis. The doctor gave me a prescription to heal my gastritis, but after taking the medicine, my illness worsened because of the spicy food that I kept constantly eating.

Use antacids to neutralize the acidic effect of gastric contents. Fortunately there are a lot of natural things that act as antacids such as almonds, bananas, milk, chewing gum, cinnamon, peppermint oil, and water

Nutritionally, there are practices which may promote the healing and prevention of gastric ulcers. Providing a more natural management practice can reduce gastric ulceration by reducing stress and allowing for a more natural feed intake process. Increased grazing through pasture turnout is recommended as the most effective dietary treatment. Grazing will allow for a more continuous saliva