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A hiker on Mount Everest exhausts their food. In these low oxygen conditions, with no food, the hiker runs out of glucose and ketone bodies important for energy production in neurons, and hence produces lower levels of ATP. 1. Of the following proteins, which protein's function is most directly/likely to be affected and why? (Ligand-gated Na' / K' channels, Ligand-gates CT channels, Na' /K", Voltage-gated Na channels, Ligand-gated K' channels) 2. Would the resting membrane potential be closer to or further away from the threshold voltage? Or, would there be no change in resting membrane potential? Please briefly explain your answer. 3. Compared to normal conditions, is this hiker more likely, less likely, or is there no change in likelihood to generate an action potential in response to the same excitatory input? 4. How would the action potential change? Below, we have a diagram of a normal action potential. Please draw any qualitative differences in the action potential (and the resting membrane potential), and explain your answer. 5. The hiker finds candy bars at a base camp and replenishes their glucose stores. ATP is now produced at normal levels again, and any previous changes to the action potential are fully reversed (the action potential is completely normal again). However, the candy bars contained a toxin called Blacker toxin, which causes voltage-gated Na+ channels to inactivate more slowly. How would the action potential change? Below we have a diagram of a normal action potential. Please draw any qualitative differences in the action potential (and the resting membrane potential) and explain your answer.

A hiker on Mount Everest exhausts their food. In these low oxygen conditions, with no food, the hiker runs out of glucose and ketone bodies important for energy production in neurons, and hence produces lower levels of ATP. 1. Of the following proteins, which protein's function is most directly/likely to be affected and why? (Ligand-gated Na' / K' channels, Ligand-gates CT channels, Na' /K", Voltage-gated Na channels, Ligand-gated K' channels) 2. Would the resting membrane potential be closer to or further away from the threshold voltage? Or, would there be no change in resting membrane potential? Please briefly explain your answer. 3. Compared to normal conditions, is this hiker more likely, less likely, or is there no change in likelihood to generate an action potential in response to the same excitatory input? 4. How would the action potential change? Below, we have a diagram of a normal action potential. Please draw any qualitative differences in the action potential (and the resting membrane potential), and explain your answer. 5. The hiker finds candy bars at a base camp and replenishes their glucose stores. ATP is now produced at normal levels again, and any previous changes to the action potential are fully reversed (the action potential is completely normal again). However, the candy bars contained a toxin called Blacker toxin, which causes voltage-gated Na+ channels to inactivate more slowly. How would the action potential change? Below we have a diagram of a normal action potential. Please draw any qualitative differences in the action potential (and the resting membrane potential) and explain your answer.

Human Anatomy & Physiology (11th Edition)
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
Section: Chapter Questions
Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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please help me to draw the plot in part 4 and 5 and explain

A hiker on Mount Everest exhausts their food. In these low oxygen conditions, with no food, the hiker runs out of glucose and ketone bodies important for energy production in neurons, and hence produces lower levels of ATP. 1. Of the following proteins, which protein's function is most directly/likely to be affected and why? (Ligand-gated Na' / K' channels, Ligand-gates CT channels, Na' /K", Voltage-gated Na channels, Ligand-gated K' channels) 2. Would the resting membrane potential be closer to or further away from the threshold voltage? Or, would there be no change in resting membrane potential? Please briefly explain your answer. 3. Compared to normal conditions, is this hiker more likely, less likely, or is there no change in likelihood to generate an action potential in response to the same excitatory input? 4. How would the action potential change? Below, we have a diagram of a normal action potential. Please draw any qualitative differences in the action potential (and the resting membrane potential), and explain your answer. 5. The hiker finds candy bars at a base camp and replenishes their glucose stores. ATP is now produced at normal levels again, and any previous changes to the action potential are fully reversed (the action potential is completely normal again). However, the candy bars contained a toxin called Blacker toxin, which causes voltage-gated Na+ channels to inactivate more slowly. How would the action potential change? Below we have a diagram of a normal action potential. Please draw any qualitative differences in the action potential (and the resting membrane potential) and explain your answer.
Transcribed Image Text:A hiker on Mount Everest exhausts their food. In these low oxygen conditions, with no food, the hiker runs out of glucose and ketone bodies important for energy production in neurons, and hence produces lower levels of ATP. 1. Of the following proteins, which protein's function is most directly/likely to be affected and why? (Ligand-gated Na' / K' channels, Ligand-gates CT channels, Na' /K", Voltage-gated Na channels, Ligand-gated K' channels) 2. Would the resting membrane potential be closer to or further away from the threshold voltage? Or, would there be no change in resting membrane potential? Please briefly explain your answer. 3. Compared to normal conditions, is this hiker more likely, less likely, or is there no change in likelihood to generate an action potential in response to the same excitatory input? 4. How would the action potential change? Below, we have a diagram of a normal action potential. Please draw any qualitative differences in the action potential (and the resting membrane potential), and explain your answer. 5. The hiker finds candy bars at a base camp and replenishes their glucose stores. ATP is now produced at normal levels again, and any previous changes to the action potential are fully reversed (the action potential is completely normal again). However, the candy bars contained a toxin called Blacker toxin, which causes voltage-gated Na+ channels to inactivate more slowly. How would the action potential change? Below we have a diagram of a normal action potential. Please draw any qualitative differences in the action potential (and the resting membrane potential) and explain your answer.
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