- What is a source and function of erythropoietin? Explain: What is meant by polycythemia? What causes polycythemia? How polycythemia would affect the value of hematocrit?
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- Describe the Molecular Mechanism of Hypertrophic cardiomyopathy: --> Normal molecular physiology and/or biochemistry: describe the relevant biochemical structures, cellular organelles, 2nd messengers, DNA/RNA, etc. related to Hypertrophic cardiomyopathy Explained clearly using your own words and provide any citation used, please.Font Ad 11 Heart Failure MI of left ventricle fatigue and exercise intolerance orthopnea hemoptysis secondary polycythemia daytime oliguria 11 left heart failure || || Paragraph Instructions: Highlight (use paint bucket) the etiologies (cause) and manifestations below as being either related to right heart failure (blue), left heart failure (red), or both (purple). Next, after each item put a (e) if it is the etiology (cause), a (m) if it is a manifestation, and a (c) if it is a manifestation that is also a compensation. One has been done as an example. Right Heart Failure Both Right and Left-Sided Heart Failure pulmonary valve stenosis rales Fi ascites edema of feet, legs and ankles tachycardia jugular vein distension hepatosplenomegaly Styles Drawing Left Heart Failure COPD systemic hypertension (e) pulmonary edema cough pallor (m) peripheral vasoconstriction aortic valve stenosis cold intolerance Select v EditingDescribe the pathogenesis of erythroblastosis fetalis including relevant maternal, baby, and paternal blood types. Why is the first pregnancy is USUALLY not a problem but each subsequent pregnancy is - remember antibody types produced. If this is the patient's first known pregnancy, how could this baby still be at potential risk should mom have the blood type that is of concern in the first place? Also describe how the fetus is affected - which system(s) are in greatest danger. Lastly, what is Rhogam and how does it work? If Rhogam fails, what other treatments may be tried?
- give examples of 4 different values of hematocrit and explain their meaningExplain the biochemical basis and clinical implications of atherosclerosis with molecular and mechanistic detail.Discuss hematocrit and hemoglobin lab tests and their significance in the diagnosis of the more common types of anemia. How can liver dysfunction cause bleeding disorder?
- define hematocrit. what is a typical hematocritA. Calculate the final percent concentration of the erythrocytes in each well: (NaCl, CaCl2, MgCl2) each solution has a concentration of 0.25M erythrocyte suspension solution (1mL of erythrocyte solution containing 10% washed red blood cells.) 135 microliters of each solution were added to individual wells + 15uL of the erythrocyte solution ex: Well #1= 135uL of (0.25M CaCl2 + 15uL erythrocyte sol.) B. What would be the final concentration of a well with 135uL (0.125M NaCl) + 15 uL erythrocyte solution?State at least five (5) conditions associated with pathologic leukopenia.
- although there are other ways to prevent DVT, do you think it's vital to advocate and require the usage of sequential compression devices to maintain blood flow and prevent blood clots? pls include references.Select all the options below which are false of hemostasis. | aspirin is an effective "blood thinner" (anticoagulant) because it prevents the formation and release of ADP by platelet plugs | after damage to a vessel, the immediate reaction is vasodilation to bring more white blood cells in and prevent infection O factor X is the converging point of both the intrinsic and extrinsic pathways of the clotting cascade when platelets begin to aggregate, a negative feedback loop begins to cause more platelets to aggregateWrite the List three types of clotting problems.