Select all of the statements that are true regarding prions O Prions are infectious nuclelc acids. O . Normal PrP proteins have alpha-hellices, while infectious PrP proteins have beta pleated sheets. Prions are associated with many tissue types. Prions are diagnosed with a simple blood test. O Prions cause diseases known as spongiform encephalopathles Prions are living acellular entities Prions are misfolded proteins which can cause normal versions to also misfold. 0000000

Select all of the statements that are true regarding prions O Prions are infectious nuclelc acids. O . Normal PrP proteins have alpha-hellices, while infectious PrP proteins have beta pleated sheets. Prions are associated with many tissue types. Prions are diagnosed with a simple blood test. O Prions cause diseases known as spongiform encephalopathles Prions are living acellular entities Prions are misfolded proteins which can cause normal versions to also misfold. 0000000

Biology 2e

2nd Edition

ISBN:9781947172517

Author:Matthew Douglas, Jung Choi, Mary Ann Clark

Publisher:Matthew Douglas, Jung Choi, Mary Ann Clark

Chapter21: Viruses

Section: Chapter Questions

Problem 9RQ: Which statement is not true of viral replication? A lysogenic cycle kills the host cell There are...

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The following statement(s) is/are .6 * correct about bacteriophages exiting 1 E) Virions of bacteriophages exit by lysing bacterial cells Bacteriophages may break down cell wall's peptidoglycan by producing enzymes Bacteriophages may inhibit host enzymes with roles in cell wall synthesis Bacteriophages lyse bacterial cells by covering their virions with cell membranes a, b, and c a, and b
Regular herpesvirus-mediated cold sores or genital sore flare-ups are caused by reinfection by the same herpesvirus strain reinfection by a closely related herpesvirus of a different strain copies of the herpesvirus genome permanently maintained in host cell cytoplasm copies of the herpesvirus genome permanently maintained in host nuclei
Which of the following is not associated with prions? Replicating shapes Mad cow disease DNA Toxic proteins
There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (vCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (vCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes vCJD. In spite of these numbers, the death rate from vCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. If you were traveling in Europe, would you eat beef? Give sound reasons why or why not.
There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (vCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (vCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes vCJD. In spite of these numbers, the death rate from vCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. What measures have been taken to stop BSE?
Which statement is not true of viral replication? A lysogenic cycle kills the host cell There are six basic steps in the viral replication cycle Viral replication does not affect host cell function Newly released virions can infect adjacent cells.
Choose all the true statements regarding coronavirus proteins. Vaccine preventable diseases include COVID-19. For coronaviruses, it is more likely to find evolving mutations in the portion of the spike protein than the nucleocapsid protein. Mutations in the genetic code for the spike protein will produce 3D changes that are more likely to affect vaccine efficacy than changes to the nucleocapsid protein code. The nucleocapsid protein interacts with viral nucleic acid specifically whereas the spike protein interacts with the host protein non-specifically.
CAN Corynebacterium diphtheriae be infected by a viruses. I know it is a bacteria but I need to know if it is possible for it to be infected by a virus. Please be specific but in terms that is easy to understand. PLEASE answer this specif question. I don't need to know the causes, effects, outcomes, etc of Corynebacterium diphtheriae. I already know that stuff, I need this specific question answered. THANK YOU.
viruses:1. Why must primary cell cultures be restarted every so often when preparing primary cell cultures to observe morphological changes caused by cells infected by a virus? Why are tumor cells preferred? 2. Why are non-enveloped viruses generally more resistant to disinfectants than are enveloped viruses? 3. A public health physician isolated large number of phages from rivers used as a source of drinking water in western Africa. They physician is very concerned that humans might become ill from drinking this water, although she knows that the phages specifically attack bacteria. Why is she concerned?
Polyomavirus and Papillomavirus, which is NOT correct? are circular dsDNA are icosahedral capsid are non-enveloped contain DNA and RNA polymerase their fusion with the host cell doesn’t involve in uncoating genome replicate in cytoplasm None of the above Both D and F D, E, and F
false true Viruses do not reproduce .within host cell Phagocytosis is type of Lactive transport R.B.Cs have no mitochondria Phospholipids have similar structure to fat Prokaryotic cell has no mitochondria Channel protein form a small opening through the cell membrane All nucleic acids have no oxygen In prophase stage chromosomes become shorter Cell is defined as basic functional unit of living .organism Phospholipid bilayer of cell membrane has hydrophilic head only O O
Can you help? I cannot seem to figure out all of the correct answers. I know lysis and budding is one but what is the other (s)? I am so lost! Thanks! Can Egress by viruses from the host cell occur by lysis or budding? Does egress by viruses immediately kill it? Does egress by viruses from the host cell happen after replication of its protein and genetic components and before assembly into new viruses? Is egress by virus from the host cell part of either lytic or lysogenic cycles?