In the past, it was believed that lactic acid accumulated in the muscles of tracking athletes as a result of intense exertion. For this reason, coaches would have athletes slowly jog to move more oxygen into the muscles so lactic acid would be oxidized and there would be less soreness the next day. Based on current research, what is the most likely consequence of lactate production in the athlete’s performance? What is more likely to be the cause of muscle soreness?

In the past, it was believed that lactic acid accumulated in the muscles of tracking athletes as a result of intense exertion. For this reason, coaches would have athletes slowly jog to move more oxygen into the muscles so lactic acid would be oxidized and there would be less soreness the next day. Based on current research, what is the most likely consequence of lactate production in the athlete’s performance? What is more likely to be the cause of muscle soreness?

Body Structures & Functions

12th Edition

ISBN:9781285695495

Author:Scott

Publisher:Scott

Chapter7: Muscular System

Section: Chapter Questions

Problem 9RQ

See similar textbooks

Similar questions

Describe the two mechanisms by which muscle glycogen is broken down to glucose (glycogenolysis) for use in glycolysis. Which one is activated at the same time as muscle contraction?
In order to study and understand muscle diseases, one needs to have a good understanding of normal muscle function. Briefly outline the process of cross-bridge cycling in muscle contraction. Be sure to highlight the role of Ca2+ and ATP/ADP in the process.
Muscle fatigue occurs because of.. A relative deficit of ATP Excessive accumulation of lactic acid Ionic imbalances Depletion of nutient stores All of the above
The filaments of skeletal muscle are moved by free energy derived from the hydrolysis of ATP. During sustained muscle contraction ATP levels remain fairly constant, while levels of creatine phosphate fall. Explain why this is so. Under what circumstances is an oxygen debt incurred during muscle contraction?
If you were able to control fatigue in the muscle cell experimentally such that you only exposed the muscle to high levels of Pi and no other metabolites, what would you observe? A decline in force without much change in velocity A decline in velocity without much change in force A muscle operating below Lo A decline in both force and velocity An increased affinity of Ca2+ for troponin
What is the normal end product of anaerobic exercise in muscle?
Which of these is the proper sequence of these events of muscle contraction 1) Action potential propagating down into the T-tubule 2) Power stroke 3) Ca+2 is released from the sarcoplasmic reticulum 4) The tropomyosin shifts out of the way thereby exposing the active sites of the actin Question 7 options: a) 1 à 3 à 4 à 2 b) 4 à 3 à 2 à 1 c) 1 à 4 à 2 à 3 d) 3 à 1 à 4 à 2
Impairment of intracellular calcium homeostasis in muscle fibers lead to chronic, severe muscle dysfunction. Impaired Calcium ATPase activity can lead to abnormal, persistent elevation of intracellular calcium concentrations. Explain how persistently high intracellular calcium concentration would affect cross-bridge formation, and muscle contractions, and muscle tension? (Mention the effects on the steps involved cross bridge formation, and muscle contraction/relaxation cycle, and overall muscle tension).
Which of the following statements is true about the control of muscle glycogen phosphorylase? a) It is activated by phosphorylation by an active phosphorylase kinase b) It is allosterically activated by ATP c) It is allosterically activated by CAMP d) Normally it exists in active form
Describe the changes that occur in skeletal muscle as a result of endurance training. Include a brief discussion on how endurance training improves acid-base balance during exercise. What physiological benefits may happen for an older adult who begins a progressive resistance-training program?
Contraction of both skeletal and smooth muscle is triggered by an increase in cytosolic Ca2+. Compare the mechanisms by which each type of muscle converts a rise in Ca2+ concentration into contraction.
Match the following regarding muscle contractions: The final chemical messenger and "trigger" for muscle contraction. It binds to troponin A neurotransmitter released at motor end plates by the axon terminals It diffuses across the cell membrane resulting in depolarization Activates synaptic vesicles in axon terminals to fuse with plasma membrane of axon terminal Used to convert ADP to ATP by transfer of a high-energy phosphate group. A reserve high-energy compound Breaks down ACh into its building blocks, rendering it ineffective [Choose] [Choose] [Choose] [Choose] [Choose] [Choose ]