In relation to each cue, describe the pathophysiology of diabetic ketoacidosis (DKA). Fruity and sweet-smelling Odor to the breath Oliguria (low urine output) Frequent urination High blood glucose levels Hypertension Excessive thirst Nausea and vomiting Fatigue and lethargy
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In relation to each cue, describe the pathophysiology of diabetic ketoacidosis (DKA).
- Fruity and sweet-smelling Odor to the breath
- Oliguria (low urine output)
- Frequent urination
- High blood glucose levels
- Hypertension
- Excessive thirst
- Nausea and vomiting
- Fatigue and lethargy
Step by step
Solved in 3 steps
- Give a brief pathophysiology of diabetic kito acidosis.Including the signs and symptoms of DKAHow does Type-2 Diabetes occur? Explain the pathophysiology and give its laboratory diagnosisdescribe the pathophysiology of diabetic ketoacidosis and explain the rationale of High respiratory rate, high temperature and high heart rate in patients with DKA List 5 key goals of nursing care associated with DKA
- Which of the following medications, when taken prior to eating, is especially effective for correcting postprandial hyperglycemia after a high-carbohydrate meal, so that hypoglycemia post dosing is minimized? Acarbose Glyburide Glitazone TolbutamideExplain the reasons why each of these signs and symptoms happen in Hyperthyroidism and Hypothyroidism. Hyperthyroidism Hypothyroidism Weight loss Weight gain Increases appetite Decreased appetite Increased sensitivity to heat Increased sensitivity to cold Diarrhea Constipation Tachycardia Bradycardia Increased hair and nail growth Hair loss and thin nails Anxiety and irritability Depression Tremors Periorbital puffiness Muscle weakness Deafnesspathophysiology Categorize the clinical manifestations and diagnostic lab values most associated with hyperosmolar coma vs. diabetic ketoacidosis Answer options: Decreased level of consciousness caused by osmotic cell shriveling Insulin is low Ketoacids Decreased level of consciousness caused by decreased pH Blood glucose >600 mg/dL Insulin is absent Blood glucose of 300 to 600 mg/dL No ketoacids
- e. Given her recent diagnosis, Julia finds it difficult to administer an appropriate amount of insulin. Before every meal, Julia carries out the following procedure. 1. Measures her blood glucose level 2. Estimates the mass of glucose in her meal 3. Injects the quantity of insulin required to metabolise the amount of glucose in her meal. She then measures her blood glucose level again two hours after her meal. Julia records all this information on an app on her smartphone. The chart below was obtained from the app on her smartphone after 24 hours. ii. Concentration (mmol/L) i. 30 25 20 15 5 0 10 Blood glucose before eating Insulin injected Blood glucose two hours after eating Breakfast Lunch Dinner Dessert Identify the meal estimated by Julia to contain the lowest amount of glucose and give a reason. Suggest two reasons why Julia's blood glucose level was 15 mmol/L in two hours after dinner.One of the indications for prescribing nateglinide is:A. Depletion of pancreatic beta-cellsB. Significant postprandial hyperglycemiaC. Tendency to develop lactic acidosisD. Resistance to sulfonylureasE. Insulin resistancediscuss how Diabetic Ketoacidosis (DKA) develops as a complication of DM II
- Discuss the role of carbohydrates in managing patients diagnosed with Diabetes mellitusescribe some of the general effects of ibotenic acid/muscimol poisoning and list the two main species responsiblefor this type of poisoning. Describe the effects of coprine poisoning and relate this to alcohol consumption. Describe the effects of psilocybin poisoning. Describe the effects of poisoning with gastrointestinal irritantsDescribe the common degenerative effects of diabetesmellitus.