exaplin the intracellular mehcanims of smooth msucle via agonist such as Hitamine and acytycholine. link hisamine to muscarnic recptors link acetycholine to a neurotransmittors Give a diagram for each.
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exaplin the intracellular mehcanims of smooth msucle via agonist such as Hitamine and acytycholine.
link hisamine to muscarnic recptors
link acetycholine to a neurotransmittors
Give a diagram for each.
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- Matching (may not use all choices; may use some choices more than once) 45. Sección Release of inhibitory neurotransmitter 46. Decreased concentration of leaky sodium channels in the membrane Arsenic poisoning prevents mitochondria from making ATP 47. 48. Extreme sodium deficiency 49. Acetylcholinesterase concentration is decreased 50. Overdose of Potassium Chloride 51. Increased concentration of leaky potassium channels in the membrane Salto de columna A. Makes RMP more negative B. Makes RMP less negativeDepending on the size and complexity of the molecule, small chemical alterations can impart significant activity differences, especially if a certain part of the molecule is critical for binding. Considering this, select structure(s) below that are expected to have high cholinergic agonist activity based on what we leamed about acetylcholine's SAR.Explain phenomenon of constitutive activity, and how inverse agonists produce their effect.
- Distinguish between acetylcholine, an acetylcholine receptor, and acetylcholinesterase. State where each is foundand describe the function it serves.Describe the activation of the β2 (beta2) adrenergic receptor, and briefly explain how this leads to increased breakdown of glycogen stores in muscle.Give an account of signalling at a neuromuscular junction through the nicotinic acetylcholine receptor.
- Shown are different derivatives of acetylcholine. A H2N H2N D 1. Which one is expected to lose its ability to bind to the receptor but could elicit CNS side effects? Explain. 2. Which is/are expected to resist metabolism by choline esterase? 3. Which one is not expected to bind and yet prone to metabolism? Explain ZIStrychnine blocks receptor sites for inhibitory neurotransmitter substances in the CNS. Explain how strychnine can produce tetanus inskeletal muscles.Caffeine prolongs the life span of acetylcholine molecules in the motor junctions. Explain how this fact is related to caffeine’s tendency to cause jitters, such as hand tremors and other small involuntary contractions.
- Describe how presynaptic targets can be regulated to affect neurotransmitter release at the mammalian neuromuscular junction.Illustrate how and when histamine occur as a neurotransmitter.What is the structue of dopamin, and suggest the binding interactions of dopamine with the receptor. Draw a suggested model for the required interactions for agonistic and antagonistic activty in dopamine receptor.