Consider a liver cell carrying out the oxidation of glucose and fatty acids under aerobicconditions. Then, a very potent and specific inhibitor of the mitochondrial ATPsynthase is added, completely inhibiting this enzyme. What will happen to the rate ofthe citric acid cycle?No change in the rate of the citric acid cycleO Increased due to the demand for reduced electron carriers to power reactions instead of ATPO Increased due to an accumulation of protons in the mitochondrial intermembrane spaceReduced due to the accumulation of the products of the cycle that act as feedback inhibitorsO Reduced as some enzymes of the cycle share the same active site as ATP synthase and willtherefore be inhibited

ATP is the energy currency of the cell and is primarily produced by ATP synthase present in the…

Answered: Consider a liver cell carrying out the…

Biology: The Dynamic Science (MindTap Course List)

4th Edition

ISBN:9781305389892

Author:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Publisher:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Chapter7: Cellular Respiration: Harvesting Chemical Energy

Section: Chapter Questions

Problem 4TYK

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Using the answer code on the right, indicate which form of energy production is being described: 1. takes place in the mitochondrial matrix 2. produces H2O as a by-product 3. results in a rich yield of ATP 4. takes place in the cytosol 5. processes acetyl-CoA 6. takes place in the mitochondrial innermembrane cristae 7. converts glucose into two pyruvate molecules 8. uses molecular oxygen 9. accomplished by the electron transport system and ATP synthase (a) glycolysis (b) citric acid cycle (c) oxidative phosphorylation
Which of the following statements about oxidative phosphorylation is correct? O H+ ions are transferred from Complex I or Complex II to ATP synthase where ATP production occ O Proton pumps transfer electrons from the cytosol to the mitochondrial matrix as electrons are tra O The chemical and electrical gradient is established between the intermembrane space and the ma electron carriers. O ATP synthase pumps electrons back to the intermembrane space as a consequence of electrocher mitochondrial matrix. • Previous
Cyanide is a toxic substance. Cyanide is a cytochrome coxidase enzyme that is competitive. The last enzyme of the electron transport chain in mitochondria is cytochrome c oxidase. One of the most common substrates for cytochrome c oxidase is oxygen. Would an increase in the concentration of oxygen affect the inhibitioncaused by cyanide? How would conditions in the mitochondrial intermembrane spacechange if cytochrome c oxidase and the electron transport chain are notfunctioning properly. .Explain why this change in conditions will be harmful to a cell.
If there is ample oxygen available, how many ATP can be produced from complete oxidation of glucose (from glycolysis up to ETS)? Assumption: 1 NADH = 3 ATP; 1 FADH2 = 2 ATP
Complex II of electron transport chain is involved in the KREB'S Cycle during the conversion of succinate to fumarate?true or false all enzymes of the citric acid cycle are located in the cytoplasm except succinate dehydrogenase which is bound to the inner mitochondrial membrne. true of false glucagon is a hormone that activates glycogenolysis-true or false high concentration of NADPH increases the rate of the pentose phosphate pathway by stimulating glucose 6-phosphate dehydrogenase- true or false
In the presence of excess oxygen, a complete oxidation of seven molecules of glucose into carbon dioxide and water, by a yeast cell, would produce approximately ATP molecules via oxidative phosphorylation only. (Consider NADH = 3 ATP and FADH2 = 2 ATP)
Cyanide is a poisonous substance. Cyanide is a cytochrome c oxidase inhibitor that is competitive. The last enzyme of the electron transport chain in mitochondria is cytochrome c oxidase. One of the most common substrates for cytochrome c oxidase is oxygen.Would an increase in the concentration of oxygen affect the inhibitioncaused by cyanide?How would conditions in the mitochondrial intermembrane spacechange if cytochrome c oxidase and the electron transport chain are notfunctioning properly.. Explain why this change in conditions will be harmful to a cell.
A chemical is applied to cells that allows the free flow of protons across the mitochondrial membrane. What is the effect on the proton gradient, ATP production, heat production, and the ETC during aerobic respiration?
The energy of electron transport (by the ETC) serves to move protons (hydrogen ions, or H") to the intermembrane space of the mitochondrion. How does this help the mitochondrion produce ATP? O This movement of protons sets up the electrochemical gradient that drives ATP synthesis in the mitochondrion. O The protons receive electrons from the NAD+ and FAD that were accepted in glycolysis ad the citric acid cycle O The protons are transferred to oxygen in an energy-releasing reaction O The protons pick up electrons from the electron transport chain on their way through the inner mitochondrial membrane
In normal mitochondria, which of the following is true for the rate of NADH consumption (oxidation)? Select all that apply Be very low if the ATP synthase is inhibited, but increase when an uncoupler (equalizer of H+ concentration) is added decrease if mitochondrial ADP is depleted Increase in active muscle and decrease in inactive muscle decrease when cyanide is added to prevent electron transfer through the electron transport chain
Each acetyl CoA that enters in the citric acid cycle, 2 carbon dioxide molecules are released in reactions which are coupled with the production of NADH molecules from reduction of NAD+ molecules and FADH2 is alse released by reduction of FAD+. NADH and FADH2, pass their electrons to the electron transport chain, turning back into NAD+ and FAD+ Explain further
Normally, the flow of electrons to O2 is tightlylinked to the production of ATP via the electrochemicalgradient. If ATP synthase is inhibited, for example, elec-trons do not flow down the electron-transport chain andrespiration ceases. Since the 1940s, several substances—such as 2,4-dinitrophenol—have been known to uncou-ple electron flow from ATP synthesis. Dinitrophenol wasonce prescribed as a diet drug to aid in weight loss. Howwould an uncoupler of oxidative phosphorylation pro-mote weight loss? Why do you suppose dinitrophenol isno longer prescribed?

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