Read in eTextbook - Chapter 19 page 405 - Replicative Cycles of Animal Virusesespecially see Figure 19.8 The replicative cycle of an enveloped RNA virusAfter reading you need to do 2 things - see A and B below.A) Post an original post answering the 2 questions. using at least 6-8 sentences per answer.question 1: what are 2 good potential targets in SARS-CoV2 virus to stop it from functioning ? (HINT: Think antibodies targeting importantviral proteins)question 2: what is the Pfizer Vaccine composed of? what does it target in SARS-CoV2 virus? Can you connect it to any concept from Ch17 in your course?

SARS-COV-2 virus is responsible for the recent pandemic and unnatural number of fatalities. It is an…

an original post answering the 2 questions, using at leas question 1: what are 2 good potential targets in SARS-CoV2 virus to stop it from functioning? (HINT: Think antibodies targeting important viral proteins) per answer. question 2: what is the Pfizer Vaccine composed of? what does it target in SARS-CoV2 virus? Can you connect it to any concept from Ch 17 in your course ?

an original post answering the 2 questions, using at leas question 1: what are 2 good potential targets in SARS-CoV2 virus to stop it from functioning? (HINT: Think antibodies targeting important viral proteins) per answer. question 2: what is the Pfizer Vaccine composed of? what does it target in SARS-CoV2 virus? Can you connect it to any concept from Ch 17 in your course ?

Principles Of Pharmacology Med Assist

6th Edition

ISBN:9781337512442

Author:RICE

Publisher:RICE

Chapter16: Antifungal, Antiviral, And Immunizing Agents

Section: Chapter Questions

Problem 25RQ

See similar textbooks

Similar questions

please tell me how bacteriophage and animal viruses multiply. please add both lytic and lysogenic cycles for bacteriophage, all steps, and the outcomes of both. For animal viruses, please include all steps and briefly explain the differences between their nucleic acids. i am having a hard trouble understanding this information if you could please explain in way where a child would understand it that would be amazing
All of the following statements correctly describes the properties of Influenza A virus EXCEPT A-Viral transcription occurs in the nucleus B-Mature virion are released by budding C-Single stranded positive sense RNA virus D-Attaches to cell-surface sialic acid
of Poxviruses are unusual among DNA viruses because their replication cycles takes place entirely in the host cell cytoplasm. How can these viruses carry out genome replication and transcription of viral genes without going to the nucleus? Briefly explain. Edit View Insert Format Tools Table 20px v Paragraph v へへヘ .V8:11 EEO 1I0 & %24 9 6 n d k 4
The cell is the basic unit of all living things, and viruses which are generally not considered living but have the ability to infect cells. Describe how the structure of a virus compares to the structure of a cell. You should explain (not list) at least two similarities and two differences between features of cells and viruses including a brief explanation of the function of those structures. Explain why viruses need cells.
Describe 4 differences and 4 similarities between viral lytic and lysogenic phases. * For each difference clearly state the reason why lytic is different from lysogenic using technical biological terms.* For each similarity you can just state what is the reason(s) the 2 phases are similar. Essay Limit: 300 words Book: Biology (Campbell) 11 edition Urry. Cain. Wasserman. Minorsky. Reece
This is question is from my micro biology class. 1. OUTLINE the steps that lead to the production of proteins from the genome of a double stranded RNA virus. List any PROCESSES involved and any ENZYMES.
HPV viruses have a distinct capability to evade confrontation with the human immune system? What are the basic virus properties responsible for this capability? (Give at least 3)
Can i get a help answering this question: The case study tells you that the HIV virus exits the cell by budding, but how can you tell that just based on the structure of the HIV virus? (see attached for the case study)
Match the terms 1Genome surrounded by a protein coat 2Functional viral particle capable to infect a new host cell 3Infectious protein 4Infectious agent that needs helper virus for infection 5T4 enzyme that creates holes in the E. coli plasma membrane 6When a temperate phage stimulates their bacterial host tostart producing a toxin (e.g. cholera, shiga) 7Virus that infects bacteria 8Hand N spikes 9Virus without an envelope 10Most common and best studies virulent phage of E. coli 11Protein coat that surrounds viral genome 12:true or false : some viruses can cause cancer [ Choose ] lambda True pathogenic stimulation False Nucleocapsid lysosome Naked virus induction lysozyme Flu viruses tobacco mosaic virus lysogeny capsid Lysogenic conversion Holin Prion satellite bacteriophage (or just phage) T4 Virion Streptococcus
Predict the replication step for the novel Miovirus. This virus was determined to have a dsRNA genome Draw out your prediction. Include any proteins/enzymes the virus will need to use, bring or express to carry out the replication. Indicate where it is likely for replication to occur in the host cell.
In this part, you will describe how herpes virus simplex 1 reproduces. This includes reproduction of genetic material, any recombination of genetic material, and production of new cells or virions. the type(s) of cells infected cell entry, replication, and exit detailed description of genome replication how mutation or recombination with other viruses may occur Original Drawing should highlight and label all of the structures you described
answer doesn't have to be long i just need an explanation, thank you! The whole world right now is concerned (or they should be) about the appearance of new variants of SARS-CoV-2. Among other things, these variant viruses have mutations that change the amino acid sequence of the spike protein on the virus surface. While this is concerning, I would like to propose that there is a limit to how far these mutations can change the spike protein. Why can't there be an endless number of new mutations of this viral protein? How would these amino acid changes affect how the virus works?