A patient has an inborn mutation causing the inability to produce succinate dehydrogenase required for the oxidation of FADH2. Will this person still produce ATP? Explain your answer,
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1. A patient has an inborn mutation causing the inability to produce succinate dehydrogenase required for the oxidation of FADH2. Will this person still produce ATP? Explain your answer,
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- Name: Hendric Nduwuba DNP and Cellular Respiration Summary https://www.youtube.com/watch?v=eLtRan2qFx0 Your assignment today is to summarize in two paragraphs (8 sentences total) the effects of dinitrophenol (DNP) on the human body. Make sure you explain what DNP is, the stages of cellular respiration, and how DNP alters the metabolism of the person using it (side effects). The following vocabulary terms need to be used: cellular respiration, adenosine triphosphate, and dinitrophenol (DNP). Your objective is to describe the role of ATP in cellular respiration in order to explain how DNP affects the rate of metabolism. The first paragraph is an introduction to the summary and the second paragraph is an explanation of the main idea. You will be graded on this criteria: main idea, explanation of cellular respiration, explanation of DNP, and paragraph structure.e/0/S Anemic patient diagnosed clinically through his symptoms which are: weakness, fatigue, shortness of breath, and dizziness. The lab. results of his hemoglobin level was less than 8 g/dl. On the examination of his Red blood cells isolated from the patient showed abnormally low level of lactic acid production. A deficiency of which one of the following enzymes would be the most likely cause of this patient's anemia? * Pyruvate kinase Hexokinase All of the above Phosphofructokinase Phosphoglucose isomerase Lactate dehydrogenasePlease answer ONLY. Or chose between A or B. Thank you. --------- 1. When doing activities like marathon, weightlifting, dancing and running a race we our breathe heavily. What causes our body to breathe heavily? A. To generate more ATP by the body B. Repaying oxygen debt 2. How many H+ are allowed to pass outside the protein complexes when electron passes through them? 3. What signals the start of the Krebs cycle? A. when glycolysis produces ATPs B. when pyruvate is converted from glucose 4. Which of the following conditions does not allow the occurrence of the Krebs cycle? A. when there is the presence of carbon dioxide B. when fermentation occurs 5. How many NADH are produced in the Krebs cycle when 2 molecules of glucose enters the cycle? 6. In aerobic respiration, glucose is first converted into pyruvate molecules. What compound must it changed to in order to proceed to the next stage? A. oxaloacatate B. puruvate 7. In the energy payoff stage, how many ATP (gross)…
- Explain biochemical pathways mechanistically. Describe the β-oxidation pathway. Describe the conversion of propionyl CoA to succinyl CoA. Describe the substrate activation processes in the urea cycle (CPS 1 and arginosuccinate synthetase).Pyruvate dehydrogenase kinase is a deffected enzyme and it is the connection between the alteration of metabolism and cancer. How the structure and action of this deffective enzyme/protein are altered or essential. This may include the regulation of this protein, its rate of synthesis, importance in rapid cell growth, etc. How does this alteration lead to cancer, enhance the survival of a cancerous cell, etc.? Any pharmaceutical treatments that are in development to target this protein, along with their biochemical basis. Show the chemical structures of any drugs that are discuss as well as their mechanism of action.Which of these enzymes is needed to maintain a steady pool of methylene THF used in pyrimidine synthesis and cancer cells proliferation? Group of answer choices Phosphoglyceratedehydrogenase Glyceraldehydegehydrogenase Glycine decarboxylase Thymidylatesynthase
- In the clinical case scenario provided, match the defective enzyme in Column A with the consequence of this enzyme defect in Column B. IND OAD DH Autopsy results from individual X showed normal levels of glucose and pyruvate, very low levels of NAD+ and very high levels of NADH. Their blood oxygen levels were normal, although the cause of death was given to be a lack of oxygen. Option A B C D Defective enzyme Phosphofructokinase Pyruvate dehydrogenase Cytochrome oxidase Lactate dehydrogenase write your answer here Consequence of this enzyme defect Impaired ATP production Impaired NAD production Impaired respiration Impaired oxygen I L OORD ORD OND DORO Gwould you expect an inhibitor of lactate dehydrogenase to be an effective anti-cancer drug? Why or why not?Answer the following questions below: 1. Individuals with a thiamine-deficient diet have relatively high levels of pyruvate in their blood. Explain in biochemical terms. 2. How would a riboflavin deficiency affect the functioning of the citric acid cycle? Explain your answer.
- Multivitamin B complex are essential compounds that are used as derivatives for compounds necessary for many metabolic activities. Give 3 vitamin B compounds and explain where and how they are used along the metabolic pathway.The Warburg effect is where tumor cells preferentially catabolize glucose to lactate even in the presence of oxygen (aerobic glycolysis), whereas normal cells preferentially catabolize glucose to carbon dioxide (oxidative phosphorylation). Treatment of tumor cells with the lactate dehydrogenase inhibitor NCI-737 blocks ATP production. Which of the following statements about NCI-737 treated tumor cells are MOST correct? 1) AGglycolysis is negative 2) AGglycolysis is positive 3) AG" for glycolysis is negative 4) AG" for glycolysis is positive 5) NAD+/NADH decreases 6) NAD+/NADH increases 1, 4, 6 1, 3, 5 2, 4, 6 2, 3, 6 2, 3, 5Otto Warburg made an interesting observation in the 1930s about cancer cells using the fermentation pathway to catabolize pyruvate even when the oxygen supply seemed sufficient for oxidative phosphorylation. One modern explanation for this “Warburg Effect” is that it protects the cancer cells from damage from free radicals. Provide a biochemical rationale for this explanation.