The Metabolic Syndrome ( Ms )

Metabolic Syndrome Metabolic syndrome has an enormous impact the United States economically. Heart disease, diabetes, and obesity are all the result of metabolic syndrome and each requires a tremendous amount of health care. Hormone imbalances can be part of the cause which require medication and hormone supplements which are costly. Unhealthy bodies break down faster and which means they require treatment longer.

Liver is known as the metabolic port of entry for any endobiotic and xenobiotic substance. The anabolism and catabolism of most of the nutrients are in major performed by the liver. Carbohydrate, protein and fat metabolism by the liver is of significance as the anomalies in metabolism of these nutrients may introduce several types of metabolic syndromes. The protective effects of aqueous green tea extract have been shown on hyperglycaemia, hyperlipidaemia and liver dysfunction in diabetic and obese rat models [74]. Different nutrients combinations were tested in a long-term feeding in experimental mouse model. Regional and continental food habit and practices are very much different. Effects of Western, vegetarian, and Japanese dietary fat

Cardiovascular and metabolic diseases are a growing problem around the world today. Not only do diseases such as diabetes and heart disease affect individuals and their lifestyles, they also affect the economy, politics, education and the professional work environment. 20-26% of individuals in the United States live with heart disease, and obesity and high blood pressure compose the greatest factors for developing this disease [20]. Studies have shown a direct correlation between the amount of sedentary time and cardiovascular and metabolic disease risk, regardless of baseline measurements [20]. Thus, increase in physical activity will decrease cardiometabolic risk factors [8]. Several studies indicate a positive correlation between physical activity and decreased cardiometabolic risk markers [2,4,11,16]. However, we now know that an individual who is getting the recommended physical activity per day can still have a large amount of sedentary time.

A person’s genetic make up has a significant influence on whether the person will become obese or not. If both parents are obese, the likelihood that their children may end up being obese too is higher compared to a situation where neither of the parents is obese or where only one of the parents is obese. This is particularly so because genetics influence the way the body stores energy and how energy is used. This can be seen in the differences that have recorded in the basal metabolic rates (BMR) among groups of people who differ by age, gender and the make up of their bodies. People who have a low metabolic rate have a higher risk of becoming overweight. The genetic similarities shared by members of one family can explain why people who come form certain families end up being overweight (DeBruyne, Pinna

indicate that inherited genetic variation is an important risk factor for obesity. It was also pointed

Switching to a healthy diet to reduce or control Metabolic syndrome is no different. Avoid eating foods that are filled with empty calories and overly processed fillers. Foods that are high in salt and oil content, such as fried foods, are also better off avoided. Clean and healthy eating can easily be the answer to eliminating your risk factors and beating Metabolic syndrome.

With over 22.5% of the current U.S. population considered to be clinically obese, compared to only 14.5% in 1980, there does not seem to be a cessation of this epidemic in sight (Hill & Peters, 1998). Goran and Weisners' (2000) proposal that "... the inherently lower resting metabolic rate in women versus men is responsible for the higher adiposity rates in women..." is wanting, especially since the potentially modifiable factors of; less physical strength, less daily free-living physical activity, and lower total energy expenditure are more likely the cause of the differences in observed adiposity between men and women. Since our genetic makeup has not appreciably changed in the last twenty years, we cannot strictly attribute the explosion of obesity to genetics. As these biological causes of obesity are disproved, a focus on the environment as a reason for obesity is taking centre-stage.

On the other hand there was a cross-sectional study of 1900 morbidly obese women less than 50 years old done by Valderhaug, Hertel, Nordstrand, Dale, Hofsø, & Hjelmesæth (2015). The aim of the study was to investigate if morbidly obese women with hyperandrogenemia (HA) have higher odds of metabolic syndrome than those without HA, independent of PCOS. The result showed that obesity had a major impact on metabolic syndrome and PCOS (Valderhaug et al.,

Metabolic syndrome is an emerging diagnosis in the medical field that has effects on patient care. Becoming familiar with it and having it become a common part of patient care will, if used appropriately will increase the lives of those who have become affected with or are at risk of becoming diagnosed with metabolic syndrome. Metabolic syndrome is a constellation of risks factors of metabolic origin that are accompanied by the increased risk of cardiovascular disease and type 2 diabetes. There are five major factors to look for; out of these five a person only needs three to be diagnosis with metabolic

The metabolic syndrome is a cluster of the most dangerous heart attack risk factors: diabetes and raised fasting plasma glucose, abdominal obesity, high cholesterol and high blood pressure. Metabolic syndrome often precedes the onset of T2D and cardiovascular diseases (10, 11). Worldwide, it has been estimated that approximately one-fourth of the adult population has metabolic syndrome (10) and that the joined burden of obesity related diseases causes 2.8 million deaths annually (1).

For example, type 2 diabetes can be developed from genetics, dietary habits, and physical inactivity. M.G’s condition was brought on by all three of these factors. M.G’s older sister was said to have type 2 diabetes, and her mother had a history of general heart issues. M.G’s genetic makeup most likely did not cause her to have type 2 diabetes, but because of how she is built it does put her at risk. M.G’s dietary habits are a big contributing factor to her condition, because the more fatty tissue M.G has, the more resistant her cells will become to insulin. For example, given M.G’s diet history, she consumes 5,790 calories a day, on average, people should consume around 2,000 calories a day depending on exercise. M.G is consuming double the recommended calorie intake for the average human, therefore this is causing her to gain weight and her blood pressure to rise and increase her risk of type 2 diabetes. M.G needs to maintain a balanced diet in order to control her diabetes and weight. Based on M.G’s diet history, she is consuming more than four times the recommended intake of cholesterol (1,400 mg instead of 300 mg), six times more saturated fatty acids than the recommended (149 g instead of 24 g), and almost five times more sodium than the recommended (10,135 mg instead of 2,300 mg). These intake values show how much M.G’s eating habits

Diabetes is considered a metabolic disorder. Glucose is obtain from the food we ingest and normally insulin is release as the glucose levels rise. Insulin is produced in the pancreas by cells called Beta cells. Diabetes results from an imbalance of the hormone insulin. When Beta Cells fail to produce insulin the levels of glucose in the blood rise as the kidney is not able to filtrate the high amounts of glucose in the blood. Another cause for diabetes can be insulin resistance. Insulin resistance occurs when the body is not able to utilize the available insulin. Insulin is in charge of maintaining the proper levels of glucose in the plasma. Insulin levels are raised when the levels of glucose in the blood are high. Insulin

Coll, A. P., Farooqi, I. S., and O’Rahilly, S. The hormonal control of food intake. Cell. 129(2), 251-262 (2007).

Not just a simple hormone, but a very strong one. This hormone basically tells your brain how much fat you have, what to do with it, and also - where to store it. It really tells the brain what to do. That’s why leptin controls appetite, fat-loss, and decides whether you have fat in your belly, hips, or spread evenly all over your

According to Krause’s Food & the Nutrition Care Process (2011), one primary health risk involved with untreated morbid obesity is metabolic syndrome, which tends to occur with the progression of excessive visceral adipose tissue and low cardiorespiratory fitness.1 Mr. McKinley’s BMI classifies him as morbidly obese, and he works as an office manager and has had a total knee replacement, which may contribute to physical inactivity and therefore increases metabolic syndrome risk. Since Mr. McKinley has elevated triglycerides (245 mg/dL) and fasting glucose (145 mg/dL), and lowered high-density lipoprotein (HDL) levels (32 mg/dL), he meets the criteria for metabolic syndrome. The presence of