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- Gene Interaction and Epistasis Hair color is due to the presence of melanin. There are two types of melanin produced by melanocytes. One is eumelanin, which is responsible for black (homozygous), brown (heterozygous), and blond (recessive) hair colors. The other is pheomelanin, which is responsible for red hair color. During melanin synthesis, MCR1gene converts pheomelanin to eumelanin. However, a variant of the MCR1 gene (MCR1variant), prevents this conversion. A cross between a blond-haired mother and a red-haired father produced 100% brown-haired children. When they came of age, one child married a double heterozygote, and both were blessed with 7 children; 2 with black hair, 2 with brown hair, 1 with blond hair, and 2 with red hair. Using the 7-step method, determine the genotypes of the P1, the F1, and the F2s. BOX your answers (if handwritten) or HIGHLIGHT your answers (if encoded). STEP 1: ________________________________________________________ STEP 2:…Equalizing the Expression of X Chromosome Genes in Males and Females Individuals with an XXY genotype are sterile males. If one X is inactivated early in embryogenesis, the genotype of the individual effectively becomes XY. Why will this individual not develop as a normal male?Equalizing the Expression of X Chromosome Genes in Males and Females Males have only one X chromosome and therefore only one copy of all genes on the X chromosome. Each gene is directly expressed, thus providing the basis of hemizygosity in males. Females have two X chromosomes, but one is always inactivated. Therefore, females, like males, have only one functional copy of all the genes on the X chromosome. Again, each gene must be directly expressed. Why, then, are females not considered hemizygous, and why are they not afflicted with sex-linked recessive diseases as often as males are?
- Gene Expression and the Impact of a Mutation. Can someone help me to answer the question 8 and 9, please? 8. How has the mutation altered the polypeptide? Is the function of the hemoglobin molecule (which includes 2 ẞ-globin polypeptides and 2 a-globin polypeptides) impaired? (Read your book to learn more about sickle cell disease.) 9. What is the relationship between the genotype in this case and the individual's phenotype? asap pleaseGamete Production: in the following examples please list the possible gametes produced 1) In humans, sickle cell anemia is a disease caused by a mutation of a single gene that codes for an important protein. The allele for the normal protein is dominant (S) to the one that causes sickle cell anemia. a. What gametes would be produced by an individual who suffers from sickle cell anemia produce? b. What gamester would be produce by an individual that is a carrier of the sickle cell trait?to only 23 rounds for a woman of age 20. That is a 6.5-fold greater number of cell divisions and proportionately greater opportunity for new point mutations. Yet, on average, 20-year-old men contribute only about twice as many new point mutations to their offspring as do women. How can you explain this discrepancy?
- Protein P, normally stimulates apoptosis or cell death when activated. Consider a cell with a mutation in one allele such that protein P is always expressed and active, while the other allele of gene P is deleted. Which of the following is true for this cell? Gene P is a proto-oncogene, and the phenotype of the cell is transformed. Gene P is a proto-oncogene, and the phenotype of the cell is not transformed. Gene P is a tumor suppressor gene, and the phenotype of the cell is not transformed Gene P is a tumor suppressor gene, and the phenotype of the cell is transformed 로to a maximum of 23 rounds for a lady of 20 years. This corresponds to a 6.5-fold increase in cell divisions and a proportionately increased possibility for new point mutations. Despite this, 20-year-old males produce around twice as many new point mutations to their progeny than women do. How do you account for this discrepancy?. Mutations in an autosomal gene in humans cause aform of hemophilia called von Willebrand disease(vWD). This gene specifies a blood plasma proteincleverly called von Willebrand factor (vWF). vWFstabilizes factor VIII, a blood plasma protein specified by the wild-type hemophilia A gene. Factor VIIIis needed to form blood clots. Thus, factor VIII is rapidly destroyed in the absence of vWF.Which of the following might successfully be employed in the treatment of bleeding episodes in hemophiliac patients? Would the treatments workimmediately or only after some delay needed forprotein synthesis? Would the treatments have only ashort-term or a prolonged effect? Assume that allmutations are null (that is, the mutations result in thecomplete absence of the protein encoded by the gene)and that the plasma is cell-free.a. transfusion of plasma from normal blood into avWD patientb. transfusion of plasma from a vWD patient into adifferent vWD patientc. transfusion of plasma from a hemophilia A…
- A reciprocal translocation between chromosomes9 and __ contributes to chronic myelogenous leukemia.Why is 26 incorrectto understand the disease and how to deal with it. The process involves studying family history, cell disease was hereditary. Even though neither of them had the disease, they wanted to know Before Sam was born, his parents decided to go for genetic counseling. They knew that sickle if they had a chance of having a child with the disease. Genetic Counseling Sam's parents to have a child with sickle cell disease. That was why Sam and his brother or helped his parents to provide Sam with the earliest possible diagnosis. They understood disease and were prepared to provide Sam with the best treatments available. They beoe involved with the Children's Sickle Cell Foundation and took part in the support groups. Gene counseling prepared Sam's parents to help Sam deal with the sickle cell disease. Pedigrees used by genetic counselors often include other information about the families Ao dates of birth, and dates of death are often included. Converting family history into a pediaree the…