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The genes in your lung cells can also be found in your muscle cells.
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- Are all of your genes expressed in every cell in your body? Explain your answer and include an example in your explanation.. The mutation causing sickle-cell anemia in humans, which changes the normal T to an A in the sixth codon (substituting valine for glutamic acid), occurs in which gene of the hemoglobin family? the a-globin gene (alpha) the b-globin gene (beta) the g-globin gene (gamma) the d-globin gene (delta) the e-globin gene (epsilon)Name 2 things the ABO gene codes for besides blood type.
- You are a studying the genetic basis of horn color in unicorns. Gene C determines the color of pigment destined for the horn by converting a pink precursor into a purple pigment. C- individuals make purple pigment whereas cc individuals accumulate pink precursor. Gene D ensures the deposition of the pigment into the horn cells, such that D- individuals have a colored horn (purple or pink), whereas dd individuals have a white horn. You breed two purple horned unicorns to each other (CcDd). Assuming Gene C and Gene D are unlinked, what is the probability that the baby unicorn has a purple horn? Please express your answer to 2 decimal places (e.g. 0.69).1. A monogenic disease is a disease caused by a mutation in a single gene. For instance, sickle-cell anemia is caused by a mutation in the HBB gene, which codes for the B- globin chain of hemoglobin. The beginning of HBB is shown here: 5'-ATGGTGCACCTGACTCCTGAGGAGAAGTCTGCCGTTACT...-3' A. Translate this HBB sequence into an amino acid sequence. B. In terms of amino acids, what is the result of the sickle cell mutation, wherein the bolded red A is changed to a T? This single mutation causes hemoglobin to aggregate, causing red blood cells to deform into a sickle-like shape rather than the normal “biconcave disk" shape. C. What would happen if the bolded blue A were mutated to at T? (This is hypothetical; it's not a mutation found in sickle-cell disease.)The Xb locus on Alouatta genus, the howler monkey X chromosome has 2 alleles, b+ and b. The b+ allele causes the presence of the Xb surface protein on their granulocyte cells, while the recessive allele b which does not allow the antigen to appear. The Xb locus is at 15 centimorgans, from the GRG locus. The GRG locus produces a normal enzyme activity. Individual howler monkeys with one form of an eye condition have the grg/grg genotype, with the males grg/Y presenting the condition. A male howler monkey with the eye condition caused by this enzyme mutation and no Xb antigen has a normal female howler monkey daughter with Xb antigen. This howler monkey female daughter is expecting a little howler monkey from the mating from a male howler monkey from a different tree. A) Determine the genotype of the howler monkey daughter, from the problem. B) Determine the types and percentages of each gametes in oogenesis produced of the howler monkey daughter female, based on the map distance given.…
- what advantage is there to having one copy of the sickle cell gene and one copy of the normal geneMany different alleles of a gene may exist in a population, yet each individual within the population does not carry more than two alleles of the gene. Is this true or false? A specific DNA mutation results in the production of a nonfunctional Chloride Ion Channel protein. The wildtype (normal) DNA sequence and the mutant DNA sequence that code for the Chloride Ion Channel protein, would they be considered different alleles of the same gene?There is a benign congenital condition called Hereditary Persistence of Fetal Hemoglobin (HPFH) in which production of the fetal hemoglobin (HbF) is not switched off post-partum (after birth). It is due to a mutation in the β-globin gene cluster. People that are homozygotes for this mutation, meaning that the gene from both parents is mutated, continue to make almost exclusively HbF throughout their life rather than adult hemoglobin (HbA). HPFH homozygotes demonstrate a slight erythrocytosis, or an increase in the number or mass of their red blood cells, and consequently an increase in the concentration of HbF. Given only what you know about the oxygen saturation dynamics of HbF versus HbA, can you explain why there might be the slight erythrocytosis in HPFH homozygotes? It has been observed that the prevalence of HPFH is much higher in populations with certain hemoglobinopathic disorders such as sickle cell anemia, and is selected for in populations with a high prevalence of these…
- A/a O A"/a afa Q afa Q Unsupplemented Supplemented b A"/a offspring Unsupplemented mother Supplemented mother This question relates to the aqouti mice research discussed in the lecture. What did the researchers observe in the experimental group (AVYA x aa) that was fed diets high in methyl donors? They gained weight rapidly. Increased expression of aqouti gene. Reduced risk of chronic disease Higher percentage of yellow offspring.How would you explain gene expression? How is it that a particular genotype is actually expressed as a phenotype? I am looking for details here, including an explanation of the molecular mechanisms involved.. Mutations in an autosomal gene in humans cause aform of hemophilia called von Willebrand disease(vWD). This gene specifies a blood plasma proteincleverly called von Willebrand factor (vWF). vWFstabilizes factor VIII, a blood plasma protein specified by the wild-type hemophilia A gene. Factor VIIIis needed to form blood clots. Thus, factor VIII is rapidly destroyed in the absence of vWF.Which of the following might successfully be employed in the treatment of bleeding episodes in hemophiliac patients? Would the treatments workimmediately or only after some delay needed forprotein synthesis? Would the treatments have only ashort-term or a prolonged effect? Assume that allmutations are null (that is, the mutations result in thecomplete absence of the protein encoded by the gene)and that the plasma is cell-free.a. transfusion of plasma from normal blood into avWD patientb. transfusion of plasma from a vWD patient into adifferent vWD patientc. transfusion of plasma from a hemophilia A…
